Colon tumours are tumours occurring in the part of the colon, which are classified as benign and malignant, malignant or colon cancer, and pose a serious health threat.
1. Probable tumours: common adenomaceous carcasses, formed from the skins on the intestinal mucous membranes, most of which look like arthropods or broad-based swelling. Fluid adenomas are at high risk and tubal adenomas are relatively low. Fatty tumours consist of fatty tissue, which is soft and slow to grow. It is benign, but it is partly likely to change and requires attention. Malignant neoplasms (coloural cancer): mostly gland cancer, and the structure of cancer cell gland. The mucous gland cancer contains a large amount of mucous fluid, which is poorly prepared. Undivisive cancers have a high degree of malignity, cancer cells have no significant dichotomy and are growing and shifting rapidly.
1. Dietary factors: Long-term high lipid, high protein and low-fibrous diets are important contributing factors. Fats and proteins stimulate intestinal mucous membranes in intestinal decomposition products, with low intake of fibres resulting in slow intestinal creeping and prolonged presence of harmful substances. Processed meat, containing nitrite carcinogens, is frequently eaten, increasing the risk of morbidity. Genetic factors: About 5 – 10 per cent of colon cancer has a family genetic background. In the case of family adenomas, with associated genetic mutations, family members are at high risk of disease. Genetically insistible carcinogenic enteric cancer is also caused by genetic mutations. 3. History of intestinal diseases: Inflammatory enteropathy such as ulcer, Crone disease, irritation of chronic intestinal inflammation, increasing the incidence of cancer. Adenomas can also lead to cancer if they are not removed in time. 4. Other factors: age growth, susceptibility to genetic mutation of colon mucous membrane cells and increased morbidity among people over 50 years of age. The hormonal levels in the obese population have changed and inflammation factors have increased, increasing the risk. Type 2 diabetes patients suffer from abnormal blood sugar metabolism, which affects intestinal micro-environment and is also associated with colon cancer.
1. Change in excrement habits: is a common early symptom. Diarrhoea, constipation or both can occur. If defecation is the norm, it suddenly continues with diarrhoea or constipation, with vigilance. Distortion of the defecation pattern: decomposed, deformed, or with blood, slime. Blood colours can be red or dark, easily confused with hemorrhoids and need to be identified. 3. Abdominal pain, abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdominal abdomes: most of the pain and abdominal abdominal ache, which is not fixed. The tumour clogged the intestine cavity, which is characterized by intestine infarction, increased abdominal pain, abdominal swelling, vomiting and stop exhausting. 4. Symptoms of the whole body: progress of the condition, anaemia, wasting, inactivity, low heat. Chronic haemorrhagic anaemia due to tumours causes wasting and inefficiency due to nutritional consumption.
Diagnosis 1. Septic sub-blood testing: simple screening method to detect invisible blood in the faeces. There are multiple tests of positives. 2. colonoscopy: diagnostic gold standard. It is possible to observe directly inside the colon, to detect pathological changes in tissue activity and to identify the benign and pathological nature of the tumor. 3. Visual screening: CT and MRI can understand the tumour area, size, morphology, whether there has been an attack on the surrounding tissue or a remote transfer. It is important for the development of treatment programmes. 4. Oncology marker detection: cancer embryo antigens (CEA), sugar antigens 19 – 9 (CA19 – 9), etc., cannot be diagnosed, but treatment monitoring has a reference value, such as post-treatment rises that may indicate a relapse.
1. Surgery: the main treatment for early colon cancer. The tumours and the surrounding parts of normal tissue are removed and the area lymph nodes are cleaned. Based on the tumour, there are right half-circumcised, left half-circumcised, etc. Positive tumours, such as adenomas, can be removed from the inner lens. 2. Chemotherapy: use for mid- and late-term colon cancer, reduction of tumours prior to surgery, increase of surgical hysterectomy rate, post-operative elimination of residual cancer cells and reduction of relapse risk. Common drugs are fluorine, Osharip, etc. 3. Rehabilitation: local end-stage colon cancer, pre-surgeon reduction of tumours, post-surgeon reduction of local relapse risk. 4. Target-oriented treatment: tumours that display abnormalities for specific gene mutations or proteins. If the Baylord is one-sided, the Westerly is one-sided, the side effects are relatively small and the treatment is good.
1. Healthy diet: Eat more of high-fibre foods such as vegetables, fruits and whole grains, reducing high fat, high protein and processed meat intake. Increased dietary fibre intake, promotion of intestinal creeping and reduction of hazardous substance stay. 2. Periodic medical check-ups: People over 50 years of age, annual septic sub-blood tests, colonoscopy every 5 to 10 years. There are high-risk groups, such as family history and the history of intestinal diseases, which begin screening early and reduce the interval once a year. 3. Treatment of intestinal diseases: timely treatment of ulcer colonitis, cronosis, adenomas, etc., to reduce the risk of cancer. 4. Lifestyle adjustment: cessation of smoking and alcohol, maintenance of regularity, moderate exercise, maintenance of healthy weight and increased immunity.
colon cancer
