Head spasms are third-generation antibiotics, whose mechanism of action is mainly antibacterial by interfering with the synthesis of bacterial cell walls.
The structure and synthesis process of the bacterium cell wall, which is the key structure for maintaining the shape and integrity of the bacterium cell, is essential for the survival of the bacteria. The main component of the cell wall is aluminum, which is a complex large molecular polymer. The synthesis of pelicans is a multi-step process. First, in the cytology of bacterial cells, the precursor substances that synthesize acetamol, including N-Acetyl Lunaline (NAG) and N-Acetyl cylindric acid (NAM) and the associated pentaazine chain. These precursors were subsequently transferred outside the membrane, where they were assembled. In the assembly process, the involvement of various enzymes was involved, in which penicillin combined proteins (PBPs) played a key role. PBPs are a group of enzymes that are located on bacterial cellular membranes, including a wide range of functional enzymes, such as translucentase and glucase. The meringue enzyme is responsible for connecting the chain to form the basic skeleton of the meringue; the meringue enzyme is primarily responsible for interconnecting the pelican-side chain in the adjoining meringue chain to form a solid mesh structure.
ii. Combining head spines with penicillin in proteins (PBPs) The head spores can be combined specifically with pBPs in bacterial membranes. The combination is highly selective and kinetic, as if a precise “key” matched a particular “lock”. When the pine and PBPs are combined, changes in the spatial structure of the PBPs can occur. Since PBPs are the key enzymes in cell wall synthesis, changes in their structure can lead to inhibition of their activity.
iii. Interruption of cell wall synthesis • Repression of transmutation: transmutation enzymes in PBPs are responsible for interconnecting the peptide chain on the adjacent peptide chain. Under normal conditions, the D-Aramyl-D-Apropamate (D-Ala-D-Ala) at the end of the pelican chain reacts with the pelican side chain on the other pelican chain, with a steady network of the pelican chain. The cortex pine binds with PBPs and inhibits the activity of transcipher enzymes. This makes the interlinking process between the pelican glucose chains irregular and the mesh structure of the cell wall incomplete and solid. It is as if, during the construction of a house, the transcipher enzyme is the worker who is responsible for connecting the building components (polymal sugar chains), while the hysteria blocks the work of the “workers” and causes problems in the construction of the “house” of the bacterial cell wall. • Interruption of the Glucose Base: The Glucose Ness is also one of the PBPs, which is primarily responsible for connecting the polyglucose chain and building the basic skeleton of apricot. While the direct interference of hysteria with the GTP is relatively weak, due to the inhibition of the GTP, the mellow chain is not properly linked and may indirectly affect the normal work of GTP. GTPs require a relatively stable environment and coordination with other enzymes in the construction of the polyglucose chain. When the rotor function is destroyed, the “flow line” of the entire cell wall synthesis is disrupted, and the function of the glucose-based enzyme is not well performed, leading to problems in the synthesis and connection process of the polyglute chain.
As a result of the hysteria that interferes with the synthesis of a bacterial cell wall, a series of physiological reactions occur. First, the barrier to cytowall synthesis leads to a thinning and vulnerability of bacterial cytowalls. The weak parts of the cytological wall expand and break as the bacterial internal osmosis pressure. At the same time, in order to sustain itself, bacteria try to activate some self-remediation mechanisms and try to restore the integrity of the cell wall. However, these restoration mechanisms are often not effective in overcoming cell wall deficiencies as a result of the continued hysteria. Ultimately, bacteria are severely damaged by cell walls, which are unable to maintain the normal form and internal environment of the cells, leading to the leakage of the contents of the cells and the death of bacteria. This microbicide has led to an important antibacterial function in the treatment of infectious diseases caused by a wide range of sensitive bacteria, in particular the relatively significant antibacterial effects of the grenacella vaginal bacteria and the good antibacterial activity of part of the grenacella positive bacteria.
