Negative reaction of antibacterial drugs

Possible adverse effects in the use of antibacterial drugs are mainly of the following types:1. Manifestation: more common, in different forms, such as red spots, rubles, and wind bands, often accompanied by itching, with different probabilitys and characteristics of rashes arising from different antibacterial drugs, such as the relatively high incidence of rashes following the use of penicillin-type drugs. 2) Allergic shock: This is a very serious and life-threatening allergy response, with acute conditions, rapid respiratory difficulties, pale skin, reduced blood pressure, loss of consciousness, and a relatively typical and high level of attention for allergies caused by penicillin, which usually requires a leather test to prevent them as much as possible. 3) Reactions to seroplasia: usually 7 – 12 days after the drug has been used, which can be seen in the symptoms of heat, joint pain, skin itching, lymphoma and swollen lymphoma, often after the use of drugs such as chronic penicillin. 2. Drugs such as penicillin, headgills, etc. Beta-neamide antibacterials are relatively allergic and, in addition, sulfamide-likes can cause allergies. II. Toxicological responses1. Toxicity to the nervous system: (i) Performance: Symptoms of dizziness, sound, hearing loss, blurred vision, and peri-neurysm. For example, the use of carbamate-like drugs can cause ear toxicity at that time, lead to a decrease in hearing or even deafness, may also cause pre-court functional impairment, causing dizziness, balance disorder, etc.; chlorocin, if used in large quantities over a long period of time, may cause octic inflammation and affect vision. Related drugs: amino sugar, chloroacin, isotope (often used for anti-tuberculosis treatment and also in the context of anti-bacterial drugs) can be toxic to the nervous system. 2. Toxicity to kidneys: 1) representation: mainly in protein urine, blood urine, tube urine and kidney function decline. Medicines such as aminomal sugar, vancocin, polycactin are more likely to have kidney toxicity when the dose is too large, long or the patient’s own kidney function is poor. Related drugs: Several types of antibacterial drugs mentioned above, as well as some of the bacterium molluscs (e.g., headcarbs, which are less commonly used and have relatively high renal toxicity), may have adverse effects on the kidneys in specific cases. 3. Toxicity to the liver: 1) Performance: May cause hepatic abnormalities, e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e., e.g., e.g., e., e.g., e., e.g., e., e., e.g., e.g., d.g., d.g., d., d. For example, the long-term use of substances such as erythiocin compounds and tetracyclics can cause liver damage and interfere with normal liver metabolic functions such as. Relevant drugs: erythrin esters, tetracyclics, Lifopins (anti-tuberculosis and partly bacterial infections), etc., should be applied with attention to their potential liver toxicity. 4. Toxicity to the blood system: (i) can occur in severe conditions such as reduction of white cells, reduction of slabs, anaemia and even regenerative obstructive anaemia. Clocin can cause severe bone marrow inhibition, leading to a reduction in the whole blood cell; sulfamide-type drugs may cause abnormal manifestations of the blood system, such as particle cell reduction, in specific individuals. Related drugs: Clocin, sulfamide and some quinone-type drugs may affect the normal functioning of the blood system. III. Double infection1. Incidence mechanisms: The long-term or large-scale use of anti-bacterial drugs, especially broad-spectrum anti-bacterial drugs, can disrupt the normal balance of the fungus in the human body and allow non-predominant micro-organisms such as bacteria and fungi, which are otherwise suppressed, to breed in large numbers, thereby causing new infections. 2. Common manifestations and related pathogens: 1) oral goose scabies: mostly caused by large-scale reproduction of white pyrocolosis, manifested in the presence of white membranes on the surface of oral mucous membranes, which are difficult to erase, most often as a result of the use of broad-spectrum antibacterial drugs by relatively vulnerable persons, such as infants and young children and the elderly. 2) Physic enteritis: mainly due to excessive growth of hard-pressed cystasy, who can suffer from diarrhoea, abdominal pain, abdominal swelling, excretion of faeces in yellow and green water, or with false membranes, etc., is often associated with the use of broad-spectral antibacterial drugs such as clinicillin, third-generation cystactin. 3) Pulmonary fungi infections: When the body resistance is reduced and normal strains are suppressed, it is possible to plant and reproduce in the lung, causing symptoms of lung infections such as cough, cough, fever, respiratory difficulties, etc. IV. Gastrointestinal reaction1. Expression: 1) Disgusting, vomiting, eating disorders: This is a more common sign of gastrointestinal discomfort, with many antibacterial drugs likely to cause different levels of nausea, vomiting and eating when used, e.g., big esters (e.g. erythrin) and tetracyclics. 2) Abdominal pain, diarrhoea: This can be caused by, inter alia, drug irritation of gastrointestinal mucous membranes or changes in the normal intestinal fungus, which can be seen in some of the patients following the use of drugs such as phenolone and sulfamide, as well as in cases of severe diarrhoea, which also requires vigilance as to whether or not it is causing pseudofilmatitis in a double infection. 3) Gastrointestinal mucous membrane damage: Some drugs can lead to gastric mucosa, ulcer, etc., such as acne antiinflammation drugs combined with antibacterial drugs (especially aspirin, etc.), are more likely to occur and increase the risk of digestive haemorrhage. 2. Examples of relevant drugs: Almost all types of antibacterial drugs have the potential to cause gastrointestinal reactions, albeit to varying degrees and frequency, and some of the major types of antibacterial drugs mentioned above have received more attention in clinical applications. Other adverse effects1. Local irritation: 1) may cause inoculation pain, cortex, etc., in the case of intramural antibacterial drugs, and may cause venomitis if the inoculation is too high and the drip rate is too high, for example, in the form of a line of red wires, local pain, bruises, etc. along the veins. Drugs and conditions: Permocin potassium salt is relatively irritating and local pain is more apparent when injected in myus; some quinone-type drugs can cause venomitis if they are inverted without attention to the norms. 2. Drug heat: 1) shows that in the course of the use of antibacterial drugs, the patient has no other apparent cause of fever, the body temperature can rise to moderate or higher levels, and the body temperature can gradually return to normal after the relevant antibacterial drugs have been discontinued. Examples of relevant drugs: Multiple antibacterial drugs can cause drug heat, such as penicillin and headbacteria, which can be associated with a variety of factors, such as drug allergies and the heating of the drug itself.