Acute pancreasitis is the inflammation of the pancreas by ingestion, oedema, haemorrhagic haemorrhage and even necrosis of the pancreas as a result of various causes. Clinical conditions are characterized by acute abdominal pain, nausea, vomiting, fever and haemorrhagic insulin. The severity of the disease varies from insulin to pancreas edema, which tends to be self-restricted, pre-pregnant and also known as acute insulinitis. Insulin haemorrhagic deaths among a small number of the heavy population, often followed by infections, perimenitis and shock, and high rates of death, known as acute acute pancreas disease.
I. Acute pancreas conditions 1. Couric conditions
Cholesterol: There is a common route between the cholesterol and the cholesterol, which can block the end of the cholesterol, turn the cholesterol back into the cholesterol, activate the pancreas and cause pancreas inflammation.
Cholesterol infections: When choreous infections occur, bacteria can enter the insulin via choreography, causing incubation of pancreas tissues and, consequently, pancreas inflammation.
Cynic worms: Cynic worms can block the larvae, resulting in a lack of choreography and, consequently, inflammation of pancreas. Alcohol
Alcohol can facilitate insulinization, with large amounts of insulin being used to increase the insulin pressure, leading to glyphocyte damage.
Alcohol also produces a large amount of active oxygen, activates inflammation and further exacerbates pancreas damage. Insulin blockage
Insulin quarries, vermin, narrowness, tumours, etc. can lead to insulin blockage, increased insulin pressure, insulin discharge, and insulin influencing. 4.xiidic intestine disease
The ulcer is penetratored by a 12-intestine ball, and the vaginitis of the adjacent 12-intestine nipples can reach pancreas, causing inflammation. 5. Surgery and trauma
Pancreatic lesions, such as blunt abdominal injuries or surgical operations (e.g. ERCP surgery), can cause circulatory disorders in pancreas and cause acute pancreasitis. 6. Metabolism
High lipid and calcium haemorrhage can lead to adiposed pancreas, local ischaemic, hairy blood vessels spreading, damage to vascular walls, difficulties in insulin excretion, blockage of insulins by stones, and incubation of pancreas. 7. Infection
Infections such as mumps, influenza A, chlamydia infection and Kosage virus can be combined with acute pancreas. 8. Drugs
Certain drugs, such as sulfur, thiomers, estrogen, etc., may also lead to pancreas. Other factors
Overeating, self-immunological diseases (e.g., dry syndrome, cholesterol, etc.) can also lead to pancreasitis.
Diagnosis
The diagnosis of acute pancreas is usually based on the patient ‘ s symptoms, signs and laboratory and image tests. Common methods include blood, urine, biochemical, abdominal B, CT etc. Through these examinations, doctors can learn about the shape, structure and functional state of pancreas and whether there are complications such as cholesterol diseases, infections, etc.
iv. Acute insulin nutritional guidance 1. The diet during the acute onset of pancreas may require the patient to fast or eat only fluid foods such as rice soup, powder, juice, etc. These foods are easily digestive, do not aggravate the condition and provide the necessary moisture and nutrition for the body. As the conditions improve, the transition to semi-fluent foods, such as fresh meals, noodles and so on, can be gradual. 2. Diet during recovery
During the recovery period for pancreas, the patient shall, under the supervision of a doctor, make dietary adjustments, as prescribed by the doctor, based on the principles of low fat, high protein, digestive, low sugar and low irritation. Vegetables and fruit can be gradually increased to supplement vitamins and cellulose.
Low-fat diet: Pancreatic inflammation patients should avoid high-fat food, as high-fat food stimulates incubation enzymes and increases inflammation. It is recommended to select low-fat foods such as coarse grains, vegetables, fruits, etc.
High-protein diet: People with pancreas are usually exposed to poor protein absorption and therefore need to increase protein intake. The choice of high-protein foods, such as tofu, eggs, fish (skinned meat, decorated chicken), which are easily digested, is recommended to help with body recovery and repair.
A small number of meals: pancreas should be avoided, with as many meals as possible per day and a small amount of food per meal, with seven cents per meal, to avoid over-stimulating pancreas and to reduce inflammation levels.
Easy to digest: Select foods that are easily digestable, such as porridge, noodles, vegetable mud, etc., to reduce the pancreas burden.
Low sugar diet: control of sugar intake to avoid excessive blood sugar fluctuations and increase pancreas burden. Food staples can be selected for low sugar, such as whole grains and rough rice.
Avoiding irritating foods: Spicy, greasy, overcooled, overheated foods stimulate pancreatic digestive enzymes and exacerbate inflammation, so food should be avoided.
At the same time, smoking is prohibited and good habits are developed.
In the light of the above, patients with pancreasitis are required, under the guidance of a doctor and a nutritionist, to perform individual dietary management in order to promote the recovery of pancreas and improve the quality of life through a scientifically sound diet.
