Progress in research into evidence of pneumoconiosis of chronic obstructive pulmonary disease in Chinese medicine

The results of the recent medical examination of chronic obstructive pulmonary pulmonary pulmonary evidence for chronic pulmonary disease in Liu Zigang.Six An City’s Second People’s Hospital are summarized in the summary of the results of the recent pulmonary geriatric certificate for chronic obstructive disease, which was analysed from the point of view of the diagnosis, the material basis, the legal regimen, etc., that the certificate should be guided by a holistic approach and the principles of evidence-based treatment, and that modern scientific methods of treatment must be applied to diagnosis and treatment, so that they can be combined with modern science on the basis of inherited traditions, so as to enrich the treatment of the present document and improve the level of treatment of this document in the medium medicine. The key words: chronic obstructive pulmonary disease; false evidence of pulmonary gas; Chinese medicine. Chronic obstructive pulmonary disease (chronic obstractive pulmonary disease, COPD) is slow, with complex causes, mainly on the basis of the chronic damage to the aerobic tract, and the mechanism of morbidity is still controversial, characterized by slow onset, long duration and increasing condition. Coughs, coughs and asthma are the main symptoms at the time of the onset of the outbreak; repeated outbreaks lead to various CPR complications. The COPD medical practitioners fall into the categories of “cough”, “panting certificate”, “pulmonary swelling”, “snaping”, “pulmonary atrophy”. Pneumonic evidence is a constant witness to the COPD medical identification. A summary of the results of the recent medical evidence against COPD pneumogas is provided below. The increasing population ageing and environmental pollution in the country, and the increasing incidence of the disease in the COPD, which is expected to be a global 3rd-degree fatal disease by 2020, are expected to be the third-degree disease [1], and the current lack of cure for the disease is particularly important for the study of the causes of the disease and for the prevention of its occurrence. The “pulmonary pulmonary mainskin” refers to skin, hairy nutrients derived from pulmonary infusion, where the strength and weakness of the human body’s resistance to the function of exorcism is determined by the strength and weakness of the pulmonary function, while the relationship between modern medicine, “pulmonary congenerate”, and pulmonary disease, which is co-epidemic, or aerobic deformity, is achieved on the material basis of “pulmonary sensification”, emphasizing the status of pulmonary ailments and the importance of pulmonary disease prevention.[3] COPD patients suffer from malnutrition in the course of their pathology, as a result of abdominal deficiency and a decline in digestive absorption, and malnutrition in the course of their pathology makes respiratory muscles vulnerable to fatigue, reduced immunity and increased acute stress, while lung gas deficiency is the underlying basis for the occurrence of malnutrition in the COPD [4]. The COPD is in the areas of “cough”, “panting certificate”, “pulmonary swelling”, “snaping”, “pulmonary atrophy” and so on. The disease is developed by chronic pulmonary disease, which is characterized by chronic pulmonary and pulmonary ailments, and by false evidence of pulmonary aerobic ailments during the stable period of the disease, which can be seen in evidence [5] as the disease develops. The magnitude of the COPD is mainly related to the pulmonary function classification, which is more in line with the pulmonary gas evidence perception [6]. It also demonstrates the importance of pulmonary retrenchment in the occurrence and development of the COP. Proof of the essence of research is the scientificization of Chinese medicine, the scientific basis for supporting Chinese medicine, and the scientificization of Chinese medicine to further refine Chinese medicine. The study has been in place for more than half a century, but it has not yet been possible to present in a scientific and complete manner the material basis of the Chinese medical theory. The COPD pneumonic evidence-based research is also a hot spot. There has also been some progress. Academics have studied changes in the soluble enzyme content in the pneumatic rinsing fluid and pulmonary slurry of patients from the COPD pneumoccultation and the behaviour of the clinical symptoms, and have concluded that changes in their solusterase content are closely related to clinical performance and can serve as an objective indicator for the evaluation of the pneumogas siltation [7]. Pulmonary arteries are a more difficult issue in the COPD pathology, with complex physiological pathologies and pulmonary aerobics as their underlying cause [8]. Some scholars believe that pneumogas hypochondriam models best fit the basic Chinese medicine theory. The pneumogas false animal model was tested for macrostatistics and microindicators, which found a significant increase in the levels of inflammatory cytogenesis such as the serum IgG in the pneumogas false evidence group rats and the reduction in the lymphocyte cell conversion rate, the cause of cancer causes, IL-6 and IL_8.[9] A significant increase in sigA, sigM levels in pulmonary plasma was observed by scholars. There have been many studies in proteomics in recent years, with scholars using proteomics techniques to test substances in pneumoglobin pulmonary tissues in rats, and significant increases [11] in model rats’ transgel protein, horned protein 23, angular protein-cell skeleton 10, glandase enzyme enzyme enzymes 1, gland carcide-related proteins, and Ras inhibited protein 1. Genetic tests of the COPD stabilization rat model by scholars revealed mutations in the mtD-NAATPase6 subgene, as well as changes in the amino acids associated with the genes, which were considered likely to result from enzymatic changes [12]. In the application of metabolic grouping techniques to the research of the COPD pneumocerobic evidence, it was found that there had been significant changes in blood amino acids in patients with the COPD pneumocerosic false testimony, including oxyglycerine, hexadecane acid, 5-phosphate, 13,14-dihydro-15 ketyl prostate E2, 3-aminopropyldehyde, N-nutan tetraethylene glycerine, β-ethyl, ethylacrylate, ethylene acid, deoxoxoxin, 8-iso-15-ketyl prostate F2, 20-carbonic acid, acetyl cellol acid, deoxynucleic acid, and 18-oxycolyte cortol, possibly the disease markers of the potential COPD pulmonary gasininoxin. [13] Among the tests of the serum of patients with pneumoceroses, scholars found significant increases in IL-8, IL-1b, TNF-α levels, and in cytological factors [14]. The COPD does not yet have a well-developed cure for treatment in Upper West China, nor does it have a reliable method of improving the condition during the stabilization period.