Prophyxia treatment: comprehensive analysis and practical guidance

Pain, a common and complex type of arthritis, causes great pain and distress to patients. Understanding the treatment of gouts is critical for the mitigation of symptoms, the prevention of onset and the improvement of the quality of life. First, basic knowledge arrhythmia is the result of an increase in levels of haematitis as a result of excessive or reduced internal urine production, and the deposition of urea acid crystals at joints and surrounding tissues, resulting in inflammatory reactions. The main symptoms include severe pain, red and red, fever of joints, which is common to large toes, ankles, knee joints, etc., and pain, which often occurs suddenly at night or in the morning, seriously affecting the patient ‘ s sleep and daily activities. Long-term hyperureaemia can also lead to the formation of aphrodisiacs, which not only affect the function of the joint, but can also be exhausting and renal, causing complications such as urea-acid kidney disease and kidney stones, which pose a serious threat to health. ii. Treatment of Autumn Spectrum: In the early use of analgesics, Autumn Phenomenon is effective in inhibiting inflammation and oscillation of inflammatory cells, reducing the response to arthritis and alleviating pain. The traditional usage is 0.5 – 1 mg per hour oral, until symptoms are reduced or there are adverse effects such as gastrointestinal discomfort (e.g., nausea, vomiting, diarrhoea), but the side effects are greater. The small-dose therapy currently recommended, i.e. the initial dose of 1 mg, followed by 0.5 mg per 1-2 hour, with a total of no more than 6 mg per day, would ensure better treatment and reduce the incidence of adverse effects. However, as a result of severe side effects such as bone marrow inhibition and liver and kidney function damage, indicators such as blood protocol, liver and kidney function need to be closely monitored during use, especially for older persons, persons with incomplete liver and kidney function, and patients who at the same time use other substances that may affect the aqueous alkali metabolism. Inflammatory drugs (NSAIDs): e.g. peso-synthesis, bichlorfonic acid, lysergic acid, etc., such drugs reduce the synthesis of prostates by inhibiting the activity of the cyclic oxidation enzymes (COX), thus acting as an antiflammation, pain relief and heat relief. The NSAIDs, which are more effective and can quickly relieve joint pain and swelling during arrhythmia, are one of the first-line drugs for the acute onset period. There are, however, some differences in the efficacy and adverse effects of different NISIDs, and appropriate drugs and doses should be selected according to the patient ‘ s specific circumstances. In general, medicines that are less gastrointestinal tract irritation and are more cardiovascularly safe should be chosen, and the principle of individualized medicine should be followed. At the same time, NSAIDs may cause adverse effects such as gastrointestinal disorders, digestive haemorrhage, damage to liver and kidney function, as well as cardiovascular events, which need to be observed during their use, and where there is a history of gastrointestinal disease, a high risk factor for cardiovascular disease and a lack of liver and kidney function, the risk of use should be carefully assessed and preventive measures, such as the use of gastric mucous membranes, may be considered. Sugar cortex hormones: Sugar cortex hormones can be used as an option when the Autumn Phenomenon and NSAIDs treatments are ineffective, patients use taboos or are unable to withstand side effects. Commonly used sugar cortex hormones, such as poignysons and capponylons, can be administered in the form of oral, muscle or arthropod injections, depending on the patient ‘ s condition and individual circumstances. Sugar cortex hormones have a strong anti-inflammation effect and can rapidly mitigate arthritis, but their long-term use can lead to a range of adverse effects, such as osteoporosis, hypertension, high blood sugar, increased risk of infection and adrenal cortex functional inhibition. As a result, sugar cortex hormones are usually used only for the short term in the period of acute onset, with a gradual reduction in detoxification after symptoms are mitigated to reduce the occurrence of adverse reactions. Care should also be taken during the use of sugar cortex hormones to replenish calcium and vitamin D, to prevent osteoporosis, and to closely monitor changes in indicators such as blood sugar, blood pressure and infection. General treatment measures for rest and braking: In cases of acute pain, the patient should minimize activity, avoid being burdened by a joint, rest in bed as much as possible, and lift up the limbs slightly above the heart level, thus contributing to the re-flow of the blood and reducing the swelling and pain of the joint. At the same time, the fixed joints may be used to avoid excessive joint activity, but a fixed period should not be too long to affect the restoration of the joint function. Local cooling: cold dressing of constricted blood vessels, reduction of local haemorrhagic and inflammatory response, and some help to relieve joint pain and swelling. An ice bag or a cold towel can be applied to a pain joint for 15 – 20 minutes at a time and can be repeated several times a day, but care is taken to avoid too long or too low a temperature to cause local freezing. III. Mitric acid treatment lifestyle intervention in diet control: A reasonable diet is essential to control blood urea levels. Patients should strictly restrict the ingestion of high-precipitation foods such as the internal organs of animals (liver, kidney, heart, brain, etc.), seafood (sardfish, phoenix, shrimp, crab, shellfish, shellfish, etc.), broth, kettle soup, beans (beans, peas, etc.) etc.; appropriate intake of foods with medium-precipitation content such as meat (cow, duck, beef, lamb, etc.), fish (grassfish, carpfish, scallop, etc.) and be careful to cook as much as possible, using low-oiled cooking methods such as water cooking, steaming and so forth; and encourage the eating of low-precipitation foods such as vegetables (except for beverages, platachine, mushrooms, etc.), fruits, whole grains, low fat or defat milk products, eggs, etc. In addition, total calorie in the diet should be controlled to avoid heavy consumption and to prevent obesity, which is one of the major risk factors for arrhythmia. At the same time, alcohol consumption, especially beer, should be reduced, as alcohol can interfere with the metabolism of urea acid, increase the generation of urea acid and reduce the excretion of urea acid, which induces ache. Adequate drinking water: multi-water can facilitate excretion of urea and reduce levels of urea. It is recommended that patients drink white water or light tea on a daily basis 2000 – 3000 ml and maintain urine at above 1500 – 2000 ml to facilitate the discharge of urine acid. The use of sugared beverages and juices is avoided, as the sugars are metabolized to urea acid in the body, increasing blood urea levels. Motivation: Appropriate exercise helps to control weight, improve body mass, increase joint flexibility and muscle strength, and is positive for the treatment and prevention of relapse. Patients can choose their own way of moving, such as walking, jogging, swimming, Tai Chi Fist, yoga, etc., with a minimum of 150 minutes per week of moderate aerobic activity. Care should be taken, however, to avoid intense and long-term arthropods, such as mountain climbing, long running, basketball, etc., in order not to induce arrhythmia or increase joint damage. In the exercise, care should be taken to supplement water and to avoid dehydration. Drug treatment inhibits the production of urea: Pyramid: It is a classic anti-urea-producing drug, which reduces the level of urea acid through competitive inhibition of yellow oxidation enzymes and reduces the synthesis of urea. The initial dose of septunol is generally 50 – 100 mg per day, 1-3 times per day, with a gradual increase based on blood urea acid levels, and the maximum dose generally does not exceed 600 mg per day. In the use of septunol, care is taken of the allergies that it may cause, and life-threatening conditions such as detached skin, virulent epilepsy, etc. may occur. Therefore, prior to the use of beryllol, HLA – B * 5801 genetic tests are recommended, and for patients who test positive, the use of beryllol should be avoided and other urea acids used to reduce the risk of allergies. In addition, it can cause adverse effects, such as gastrointestinal disorders, damage to liver and kidney function, and abnormal blood system, which require regular monitoring of indicators during treatment and, in case of abnormalities, the timely adjustment of drug doses or withdrawals. Non-Bust: He is also a yellow oxidation inhibitor, which is more ureic acid-reducing than septol and does not need to adjust the dose for patients with moderate or incomplete kidney function, and the drug interacts relatively less, with better safety and tolerance. His initial dose was generally 40 mg per day and could be adjusted to 80 mg per day or 120 mg per day at the level of blood urea. In recent years, however, studies have found that he may increase the risk of cardiovascular events, especially for patients with high risk factors for cardiovascular diseases, and that the advantages and disadvantages should therefore be fully weighed in the use of non-Buzzer and that cardiovascular-related indicators such as blood pressure, heart rate, electrocardiograms, etc. should be closely monitored. The promotion of urea acid excretion drug: benzene bromomalon: benzene bromomalon promotes urea acid excretion by inhibiting the heavy absorption of urea by the kidney tube, thereby reducing the level of urea acid. The initial dose of phenylbromomalon is typically 50 mg per day, which can gradually increase to 100 mg per day, after breakfast. During the use of phenylbromomalon, patients need a large amount of drinking water to ensure daily urine in excess of 2000ml to prevent urine from forming crystals and quarries in the urine path. At the same time, alkalin drugs such as sodium bicarbonate, alkaline urine, and preservation of urine pH values between 6.2 – 6.9 can be appropriately administered, increasing the solubility of urine acid and reducing the risk of rock formation. However, benzene bromomalone should be banned for patients with a history of urinary typhus, severe kidney incompetence (ecstasy clearance rate <20ml/min) and allergy to sulfamine-type drugs. Urate oxidation enzymes: For persons with severe pain, in particular those with a poor effect of conventional urea acid reduction medication or with a taboo certificate, treatment with ureaate oxidation may be considered. Uacid oxidation allows oxidation of urea acid to the more soluble urea cysts, which promotes excretion of urea acid and reduces blood urea levels quickly. The current common urea oxidation enzymes in the country are Labliase and Prikashi, both of which require intravenous drug injection at relatively high prices. In the use of urea-oxidation enzymes, allergies such as allergies, infusions and solubles may occur and therefore need to be closely monitored and appropriate preventive and treatment measures put in place. Targets for urea acid treatment and monitoring of treatment targets: In general, for pain-free patients, the blood-urea acid target should be controlled at <360 mmol/L; for patients with pain stones, the blood-urea acid target should be controlled at <300 mmol/L to facilitate the dissolution of the aresistence and prevent the formation of new arrhea stones. However, levels of blood urea acid should not be too low to increase the risk of neurological, cardiovascular and other diseases. Monitoring indicator: In the course of urea reduction treatment, the patient is required to periodically review the level of blood urea acid, which is generally recommended for review once a month until the level of blood urea has been stabilized, with an appropriate extension of the review interval to every 3 – 6 months. At the same time, indicators such as liver and kidney function, blood and urine routines should be regularly examined to understand the safety and effectiveness of drug treatment and to detect and address in a timely manner possible adverse medical effects and complications. In addition, for patients with aphrodisiac, periodic joint ultrasound, dual energy CT, etc., can be performed to observe changes in the size, number and location of aphrodisiacs and to evaluate the effects of the treatment. IV. Treatment of renal complications with complications: Long-term hyperureaemia can lead to kidney damage and complications such as urea acid kidney disease and kidney stones. In the case of urea-renal disease, care should be taken to protect the kidney function, to avoid the use of renal toxicity drugs and to control underlying diseases such as hypertension and diabetes, in addition to the active control of blood-ureal acid levels. In the case of renal stones, appropriate treatments, such as multi-water, alkaline urine, drug soluble, in vitro shocks, or surgery to extract stones, should be selected according to the size, location and circumstances of the patient. At the same time, kidney ultrasound, kidney function, etc. are regularly checked to monitor the progress of kidney pathologies. Cardiovascular complications: High ureaemia is closely related to the development of cardiovascular diseases, with a high risk of cardiovascular disease in persons suffering from arrhythmia. Thus, in the treatment of pain, attention should be paid to the prevention and control of cardiovascular diseases, including control of blood pressure, blood sugar, blood resin, cessation of alcohol and tobacco, maintenance of a healthy lifestyle, rational use of anti-sphyllic tablets such as aspirin and cardiovascular protection drugs such as cardiovascular acids, and reduction of the risk of cardiovascular events. Long-term management and health education gout is a chronic disease that requires long-term management and treatment. Patients should be fully informed about the mechanisms for the onset of a stroke, the treatment methods and the elements of self-management, actively cooperate with the doctor ' s treatment, comply strictly with medical advice, take medication on time and review regularly. At the same time, the maintenance of a healthy lifestyle and the avoidance of induced factors, such as cold, damp, fatigue, stress, high diets, drinking, etc., prevent the recurrence of a stroke. In addition, patients should be sensitive to their mental health, and repeated attacks of arrhythmia may cause anxiety, depression, etc., and psychological attention should be provided in a timely manner, with the help of a psychologist if necessary. In summary, the treatment of arrhythmia is a comprehensive process, which includes analgesics during acute onset, urea acid treatment during mitigation periods, management of complications and long-term management and health education. Through scientifically sound treatment and management, most persons with arrhythmia are able to effectively control their condition, mitigate symptoms, improve their quality of life and reduce the risks to their health. What needs to be noticed in the diet of a man with a 1,000-word porphyrogenesis? Recommend some authoritative medical literature on the treatment of gout.