Sphinx disease is a spiral, anaerobic, and very demanding bacteria for growth conditions, which determines its unique living environment, the stomach, which produces a special enzyme: urea enzyme, which changes the surrounding environment and adapts it to. In 1983, for the first time, it was successfully separated from the gastric mucous membrane active tissue of patients with chronic active gastroenteritis, and the fungus cysts were shown to be the cause of chronic active gastroenteritis as an experimental target. Sphinx infection includes stomach inflammation, digestive ulcer, lymphatic lymphoma. The bad prognosis of the fungus coli infection is the cause of stomach cancer. The fungus fungus fungus is parasiteed in the gastric mucous tissue, with 67-80 per cent of stomach ulcers and 95 per cent of twelve-finger ulcers caused by the fungus. The common symptoms of chronic stomachitis and digestive ulcers are: upper abdominal saturation, pain, often accompanied by other adverse symptoms such as heating, abdominal swelling, anti-acid acidism and abdominal decline. Some patients can also experience repeated abdominal abdominal pains (after stomach ulcer pains, ulcer abdominal pains, etc.), small haemorrhages in the upper digestive tract (in the form of black defecation). The clinical process of cholesterococcal infection is generally thought to be as follows: the fungus fungus enters the stomach mucous and settles the infection, causes chronic, shallow stomachitis after weeks or months, develops into twelve fingers of ulcer, stomach ulcer, lymphoma, chronic atrophy, etc., and chronic atrophy is considered pre-cancer. According to the experts, the risk of stomach cancer increased by 2.7 to 12 times as a result of cholesterococcal infections, with at least 35 to 89 per cent of stomach cancer occurring without cipococococcal infections. There is a consensus among scholars that claustrospirosis is acquired by the day after tomorrow. The most likely means of transmission is through mouth and faeces, where relatively few studies are available. Sphinx is infected in populations of different races and regions of the world and is arguably the most widespread chronic bacterial infection among adults. The overall trend is that the infection rate of cholesterol is rising with age, with about 80 per cent in developing countries, about 40 per cent in developed countries and slightly more men than women. Our infection age is about 20 years older than in developed countries, 45.4-63.6 per cent of infection rates are between the ages of 20 and 40 years, and 78.9 per cent are above the age of 70. Moreover, the rate of infection in the northern part of the country is higher than in the southern region. The higher rate of infection in the country is due to co-eating, which spreads through saliva among family members. Like other infectious diseases in digestive tracts, the key to the prevention of cholesterococcal infection is to cut the transmission route by “into the mouth”. If you wash your hands before and after eating, the diet, especially the cold food, is hygienic, it is a wise choice to have a meal split when you eat together, and if you have a claustrospirosis disease in your family, you should take a meal split for the time being until it is fully cured. Our scholars share two sets of views: some believe that the infection of the cholesterol is supposed to be eradicated, while others believe that the cipocobacteria exists as humans evolve, and that there are no corresponding symptoms that do not recommend its eradication. The former is in favour. There are a number of methods for the diagnosis of cholesterocococcal infection, such as bioorganization mirrors, the separation culture of circulosis, rapid urea enzyme tests, carbon 13 or carbon 14 excretion tests, urine ammonia excretion tests, serology tests and polypolycin chain reactions. Different hospitals use different methods, but most of them are dominated by carbon 13 exhalation tests and positive hints of infection. To warn patients, such as those who feel sick in their stomachs, whose breath stinks in their mouths and the taste of their mouths, that they should be wary of the infection of the fungus coli, and that they should go to a major hospital for an examination of the infection of the fungus coli so that they can use drugs and eradicate it at an early stage in order to prevent it from developing into a serious stomach disease. Sphinx is curable. The general treatment principle is based on antibiotics, which are supplemented by acids (e.g. Omerazole, Rembellaazole, Plasconium, etc.) and gastric muscular protection agents (e.g., if americium) and commonly used antibiotics such as metallazole, nitronitro, furanketone, Kracomosone and Amocrin. Most of them are now four combination therapies: two antibiotics + one acidicant + one gastric mucous protection agent (paragus), oral for two weeks, and after four weeks of detoxification. After review, it remained positive and two antibiotics were replaced. The latest guide recommends that drugs go to Volola, are expensive and difficult to purchase and are not recommended.
It’s gastric inflammation caused by the fungus, the fungus-related stomach ulcer, the fungus-related ulcer, the fungus-related ulcer.
