The stomach ulcer is a common digestive system disease that occurs mainly in the gastric mucous membranes, which, in serious cases, can be exhausted to the lower membranes, the muscles and even the membranes. Under normal circumstances, the gastric mucous membranes have self-protection mechanisms that are resistant to gastric acid and gastroprotease digestion. However, when certain factors undermine this balance, the gastric mucous membranes are damaged and ulcer is formed.
The symptoms of stomach ulcer are diverse. Upper abdominal pain, which is the main symptom, is mostly in the nature of innuendo, swollen, burning or severe pain, and is often rhythmical. For example, pain in stomach ulcer tends to occur some time after eating, usually begins in about half an hour to an hour after eating, gradually abating after 1-2 hours, and almost disappears before the next meal, as a result of the increase in stomach acid stifling and the stimulation of the ulcer face. In addition to pain, the patient may be associated with indigestion symptoms such as anti-acid acids, gas, nausea, vomiting, appetite loss and weight loss. Some patients may not have visible symptoms, or begin with complications such as haemorrhage, perforation, such as sudden vomiting of blood, black defecation, prompting a gastric ulcer to cause cardiac fractures of blood; severe abdominal pain and the rapid spread of pain to the whole abdomen, possibly caused by an ulcer perforation, which is more urgent and requires immediate medical attention.
The causes of stomach ulcer diseases are more complex and are mainly related to the infection of the fungus (Hp), drug factors, dietary factors, stomach acid and gastroprotease, stress and psychological factors, and genetic factors. Through its spiral structure, the fungus fungus easily drills through the gastric mucous membranes, damages the gastric mucous membranes barrier and triggers inflammation reactions, which in turn leads to stomach ulcer. The long-term use of inflammatory drugs (e.g. aspirin, Broven, etc.) and sugary cortex hormones inhibits the synthesis of prostates from the stomach mucous membranes, undermines the protective effects of the gastric mucous membranes and increases the risk of ulcer occurrence. Poor dietary habits, such as chronic high consumption of alcohol, smoking, consumption of spicy irritating food, and irregular diets, can also cause irritation and damage to stomach mucous membranes. Stomach acids and gastroproteases are the direct causes of ulcer formation, and when stomach acids are overstretched or stomach mucous membrane defences are weakened, stomach acids and gastroprotease “self-indigestion” of the gastric mucous membranes into ulcers. In addition, severe trauma, large surgeries, widespread burns, cerebral diseases, and psychological factors such as chronic stress, anxiety and depression can affect the blood circulation and genus function of the gastric mucous membranes and contribute to the ulcer of the stomach. There is a relatively high risk of ulcers in the family and genetic factors may play a role in ulcers.
Diagnosis of stomach ulcer relies mainly on stomach mirror and gastric mucous activity. The stomach lens is able to observe the inside of the stomach directly, with a clear view of the ulcer’s vertebrae, size, morphology, edge, etc., and is also able to conduct a biopsy of the suspicious pathologies, to determine the nature of the pathology, and to exclude malignant diseases such as stomach cancer. In addition, upper ingestion can also be used for stomach ulcer diagnosis, which can show stomach contours, creeping conditions and ulcers, but the diagnosis of microtransformations is less valuable than a stomach mirror. The detection of cholesterocella is also essential, and commonly used methods include carbon-13 or carbon-14 exhalation tests, rapid urea enzymes tests, etc., to determine the existence of cholesterococcal infections, with a view to developing targeted treatments.
The treatment of stomach ulcer is aimed at the elimination of causes, the mitigation of symptoms, the promotion of ulcer healing, the prevention of relapse and the avoidance of complications. For those infected with positive cholesterocella, a combination of proton pump inhibitors (e.g., Omera, Lansola, etc.), americium (e.g., potassium acetate) and antibiotics (e.g., Amosilin, Kracin, etc.) is usually used, with a general treatment of 10 – 14 days. Acid treatment is an important part of the treatment of stomach ulcer. Proton pump inhibitors or H2 receptor stressants (e.g. Renedididing, Famotiding, etc.) can inhibit stomach acid sterilisation, reduce the stimuli of stomach acid to the ulcer surface, and promote ulcer healing, usually for 4 – 8 weeks. The gastric mucous membranes (e.g., magnesium aluminium carbonate, sulfur aluminum, etc.) can form a protective membrane on the stomach mucous membrane surface to prevent the erosion of gastric acids and gastric proteinases and facilitate the recovery of ulcer. In the course of treatment, care must also be taken to adjust the way of life, to maintain regular working hours and to avoid overwork and stress; to stop smoking and alcohol; to choose food that is digestable and nutritious in diet; to avoid the consumption of stimulating foods; to regularly measure meals; and to avoid the use of excessive consumption.
If stomach ulcer is not treated in a timely and effective manner, a series of serious complications may arise. Hemorrhage is the most common complication, and hemorrhage can lead to haemorrhage, black defecation, dizziness, panic, incapacitation and other forms of haemorrhage, which can endanger life in serious cases. The perforation allows for the flow of stomach contents into the abdominal cavity, causing acute peritonealitis in the form of severe abdominal pain, abdominal stress, pressure pain, anti-jump pain, etc. and requires urgent surgical treatment. The barrier is mostly caused by a constriction of blood, oedema or oscillations around the ulcer, and the patient suffers from abdominal saturation, vomiting and food, which can lead to chronic malnutrition and hydrolytic disorders. A small number of ulcer cancers may also occur and, although the rate of cancer is low, there should be a high level of vigilance about the potential for cancer, periodic review of the gastroscope and pathological examination of those who are chronically persistent, with persistent symptoms, older and associated with signs of wasting, anaemia, etc.
The stomach ulcer is a preventable disease. In our daily lives, we should be careful to maintain good living and eating habits, actively prevent the infection of the cholesterol, and carefully use drugs that may damage the stomach mucous membranes. In the event of symptoms such as abdominal discomfort or pain, medical treatment should be provided in a timely manner, with a view to early diagnosis and treatment, prevention of complications and protection of stomach health.
