Abstract: Hypocrisy enteritis is an acute mucous depravity that occurs mainly in the colon and is often accompanied by the formation of pseudoplasms, which can lead to a range of symptoms such as diarrhoea, abdominal pain and fever, which seriously affect the health and quality of life of patients. This paper examines in depth the causes of pseudo-film enteropathy, the mechanisms for its occurrence, clinical performance, diagnostic methods, treatment strategies and preventive measures aimed at raising public awareness of the disease and promoting its effective prevention and treatment.
Introduction
Hypocrisy enteritis is not uncommon clinically, especially with the use of specific drugs such as antibiotics or immunosuppressants, and its incidence tends to increase. Because of the lack of specificity of their symptoms, they can easily be confused with other intestinal diseases, leading to misdiagnostic or non-diagnosis and delays in treatment. Therefore, a comprehensive understanding of the knowledge of pseudo-film enteritis is essential for the timely detection, accurate diagnosis and effective treatment of the disease.
II. Causes of illness
1. Antibiotic relevance: This is the most common cause of pseudo-film enteritis. The long-term or unreasonable use of broad-spectral antibiotics, such as clinicillin, aminocin, sepsis, and so forth, can disrupt the normal intestinal glucose balance, and cause a very large number of bacteria, such as hard-to-baccus, to breed and produce toxins, which in turn triggers intestinal inflammation.
2. Other factors: In addition to antibiotics, chemotherapy, immunosuppressants, etc., may also cause intestinal fungus disorders and induce pseudofilmitis. In addition, intestinal ischaemic blood, surgery, severe underlying diseases (e.g. malignant tumours, diabetes, etc.) leading to a decrease in the immune capacity of the organism, also increasing the risk of the development of pseudofilm intestine.
III. METHODOLOGY
Under normal conditions, there are large symbiotic strains in the intestinal tract, which constrain each other and maintain a microecological balance in the intestinal tract. When this balance is undermined by factors such as the use of antibiotics, bacteria that produce toxins, such as hard-to-feeds, are bred. The toxins A and B produced by the tough swarms are the main cause of pseudo-film enteritis. Poison A causes inflammation of intestinal mucular cells, causing cell variability and necrosis, while toxin B acts primarily on cell skeletons, undermining cell structure integrity. Under the effects of toxins, intestinal mucous membranes are filled with blood, oedema, decomposed and forged. Hypothetical membranes, consisting mainly of cellulose, mucous protein, dead upper skin cells and inflammatory cells, are covered by damaged intestinal mucous membranes.
IV. Clinical performance
Diarrhoeal diseases: The most prevalent symptoms of pseudo-film intestinal disease are manifested in large amounts of water, ranging from several to dozens of times a day, with severe dehydration and electrolyte disorders. In some cases, floating pseudo-film fragments are visible in the faeces, which is one of the characteristic manifestations of pseudo-film enteritis, but not all.
2. Abdominal pain: Abdominal pain, often for the lower abdominal pain or for the whole abdominal pain, may be of varying degrees. Diarrhoea is associated with a high abdominal pain, which can be mitigated by defecation.
3. Heat: Most patients can have fever, generally between 38°C and 39°C, can have low, moderate or high heat, and the degree of fever is related to the severity of the condition.
4. Other symptoms: All-body symptoms of nausea, vomiting, appetite and inactivity can also be associated. Severe pseudo-film intestinal inflammation can lead to complications such as venomous coliform, intestinal perforation and sepsis, with corresponding symptoms such as abdominal swelling, severe abdominal pain, shock, etc.
V. Diagnosis
1. Medical history inquiries: Detailed information on the recent history of the use of antibiotics, chemotherapy or immunosuppressants, as well as on the presence of intestinal surgery, as well as inducing factors such as ischaemic diseases.
2. Clinical performance: Based on typical symptoms of diarrhoea, abdominal pain, fever, especially when accompanied by large quantities of water and pseudo-film fragments, there should be a high level of suspicion of pseudo-film enteritis. However, these symptoms are not specific and need to be further examined for diagnosis.
Laboratory inspection
– Excreta testing: conventional excreta testing has seen an increase in white cells and hidden blood tests are positive. Hard-to-peat toxin detection is an important basis for the diagnosis of pseudo-film enteritis, and commonly used detection methods include ELISA testing of toxins A and B, cytotoxicity tests, among which cytological toxicity tests are more specific but relatively complex.
– Conventional blood tests: white cell counts can be increased, mainly by neutral particle cells, reflecting inflammation reactions in the organism.
4. Endoscopy: colonoscopy is an important means of diagnosing pseudofilm enteritis. Under the inner mirror, it is visible that the colon mucous membrane is full of blood, oedema, decomposed, and that the surface is covered with a yellow, white, dispersed or fusioned membrane, which can be sprawled, dotted or map-formed, and that the mucous membrane around it can be dizzled. The endoscopy allows for further clarification of the degenerative nature and the exclusion of other intestinal diseases.
5. Visual examination: An abdominal X-line examination reveals non-specific manifestations such as intestinal extension, gas and fluid, which can be of some assistance in the diagnosis of the toxic coronary. CT examinations can provide a clearer picture of pathologies such as thickening of the colon wall, oedema, permeation, etc., and help to assess the condition and detect complications.
Treatment
1. Discontinuation of the drug: Antibiotics, chemotherapy or immunosuppressants that could induce the disease should be discontinued immediately upon suspicion of pseudofilm enteritis.
2. Anti-infection treatment: In the case of hard-to-scorbic infections, the most common drugs are metastasis and vancin. Americium is generally used for oral treatment at 0.4 g per day, 3 – 4 sessions per day, 7 – 14 days of treatment, and vancomicin at 0.125 – 0.5 g per day, 4 sessions per day, 7 – 14 days of treatment. The price of mitazole is relatively low, but a small number of patients may have adverse effects such as gastrointestinal insufficiency; Vancomycin treatments are positive, but higher prices and long-term use may lead to the creation of drug-resistant strains. In the case of severely ill persons or the non-effective treatment of americium, a change of treatment to Vancomicin may be considered.
3. Supporting treatment: Patients who are prone to dehydration, electrolyte disorders and acid alkali balance disorders due to a large number of diarrhoeas should be supplied with water and electrolyte in a timely manner, which can be corrected by means of oral rehydration salts or intravenous infusion of physiological saline water, glucose, potassium chloride, etc. For those suffering from malnutrition, nutritional support should be provided to ensure adequate calorie and protein intake and promote intestinal mucous membrane restoration.
Micro-ecological agent treatment: Micro-ecological formulations, such as pyrocococcus IV, acidic emulsium capsules, etc., can be appropriately applied in conjunction with or after anti-infection treatment to regulate the intestinal community balance, promote the recovery of normal intestinal strains, and contribute to improved treatment effectiveness and prevent recurrence.
5. Treatment of complications: In cases of complications such as in virulent coliforms, intestinal perforation and sepsis, appropriate treatment should be provided in a timely manner, such as gastrointestinal repressure, surgical treatment, anti-infective shock treatment, etc.
Prevention
Reasonable use of antibiotics: strict control of signs of their use and avoidance of misuse of broad spectrum antibiotics. During the use of antibiotics, changes in the patient ‘ s condition should be closely observed and, in the event of adverse effects such as diarrhoea, should be stopped and examined in a timely manner.
2. Strengthening of hospital infection control: hospitals should strengthen the clean-up of the ward environment and strictly enforce a hand-sanitary system to prevent the transmission of pathogens such as the hard-to-work swarm. Patients diagnosed or suspected to be perfunctory should be segregated to avoid cross-infection.
3. Improving the immune capacity of the organism: for people with basic diseases and low immune capacity, the treatment of basic diseases should be actively pursued, nutritional support should be strengthened, physical exercise should be appropriate, the body ‘ s resistance should be increased and the risk of the outbreak of pseudo-film enteropathy reduced.
Conclusions
Hypocrinitis is a intestinal disease that is closely related to factors such as antibiotics, with complex mechanisms, diverse clinical performance and a combination of factors for diagnosis and treatment. Measures such as increased awareness of pseudofilm enteritis, rational use of drugs, improved prevention and control of infection, and increased immunity of the organism can effectively prevent the occurrence of pseudofilm enteritis, reduce its morbidity and mortality, and guarantee the health and quality of life of the intestinal tract of the public.
Hypocrisy.
