Syphilis arthritis: The “ureic acid crisis” of the joints. It’s like a little demon hidden in a joint, and it’s sorely painful, causing great distress to patients’ lives. Uric acid is the end product of human metabolism. Under normal conditions, urea acid is excreted through kidneys and intestinal tracts, leaving the level of urea acid in the body in its normal range. However, in the case of arrhea arthritis, there is over-production of or reduced excretion of urea, leading to higher levels of urea acid in the blood and high levels of urearate. When the concentration of urea acid in the blood exceeds its saturation, the urea acid is distilled in crystallized form and deposited in joints, skims, cartilage and surrounding tissues. These urea acid crystals, like tiny “springs”, stimulate tissue around the joints and trigger inflammation. Symptoms of arrhythmia are typical of sudden joint pains. The most commonly exhausted joint is the first toe joint, the big toe joint, and patients are often awakened by this pain at night. The degree of pain is very severe, as it is cut like a knife, a needle or bite, and the skin around the joints is red, swollen and heated and local stress is evident. At the beginning of the onset, the symptoms may be self-mitigated for days or weeks, but if the conditions are not controlled, the onset will become more frequent and the pain will continue for longer periods, and may be tired and other joints, such as ankles, knees, wrists, etc. As the condition evolves, the long-term sedimentation of urea acid crystals leads to joint malformations, affects the normal functioning of the joints and even causes disability. The diagnosis of arrhythmia is based on the patient ‘ s history, symptoms, signs and relevant laboratory examinations. Doctors ask for details about the frequency, duration, pain and so forth. During the medical examination, the focus is on the red and edema, the stress and the restricted activities of the joints. The increase in blood ureal acid levels in laboratory tests is an important basis for diagnosis, but it needs to be noted that during acute onset, normal levels of blood ureal acid may occur in some patients. In addition, if a joint test reveals a urea acid crystal, it can be diagnosed as arrhythmia. Visual examinations, such as X-rays, CTs, ultrasounds, can help doctors to observe changes in the structure of joints, to learn about the deposition of urineate and whether the joints are damaged. The treatment of pyretic arthritis is aimed at the control of acute pain and inflammation during the onset period, the reduction of levels of urea acid and the prevention of re-emergence and joint damage. During acute onset, inflammation drugs, such as Bloomen, Piming, etc., are used, which can effectively reduce pain and inflammation. Autumn pixieline can also be used if the patient has taboos or is unable to withstand anti-inflammatory drugs. For seriously ill patients, sugar cortex hormones can also be used to mitigate symptoms. In intermittent and chronic periods, treatment focuses on reducing levels of urea acid. Commonly used urea acids are those that inhibit the production of urea, such as sequinols, non-buttas, and those that promote phenylbromomalon. In the use of urea acids, care should be taken to gradually increase the dose, starting at small doses, while monitoring levels of urea acid to slow down and avoid a re-emergence due to the rapid decline in urea acid levels. Self-management of patients in their daily lives is also important for the control of arrhythmia. In terms of diet, patients are required to severely limit the intake of high-pretium foods, such as animal internal organs, seafood, meat soup, etc., and to eat more low-precipitous foods such as vegetables, fruit and whole grain. At the same time, alcohol consumption, especially beer, should be avoided, as alcohol affects the excretion of urine acid. Drink more water and at least 2,000 ml of water per day, which increases the excretion of urine acid. Maintain appropriate weight to avoid obesity, which increases the generation of and reduces the excretion of urine. Appropriate physical exercise is carried out, but care is taken to avoid overwork and joint damage, and during a gout, as much rest as possible to reduce joint activity. Although it is a more difficult disease, a person can improve the quality of life by effectively controlling the condition, reducing the frequency of onset and protecting the function of the joint through reasonable treatment and good living habits.
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