The causes and patterns of deformity in the downside of the delta.

The deformation of the tripod is a relatively rare congenital heart disease, which is usually accompanied by changes in the structure of the tripodal petals and the right-heart morphology, which can lead to dysfunctions and cardiac disorders in the right heart. The following is a brief description of the causes and clinical performance:Pathology: In the early stages of the embryo, the development of the heart is a complex and fine process, with the three-tip petals being shaped by a series of processes, such as the separation and integration of the inner-dural pads and the cardiac tissues. If the process is disturbed by internal (e.g., abnormal transmission of the embryo ‘ s own cell signal, genetic defects, etc.) or external (e.g., exposure to the rubella virus during pregnancy, megacell viruses, exposure to harmful chemicals, certain degenerative drugs, etc.) factors, it can easily lead to a development error in the three-pores, and the lack of normal formation and positioning of the leaves and their subsidiary structures, leading to a deformation.• Genetic factors: Some patients have family genetic background, certain genetic mutations or anomalies may be related to the occurrence of sub-tripoly deformations, although the exact genes that cause the disease are not yet fully known and the relative complexity of the genetic patterns of the disease within the family may present different genetic patterns.Clinical performance varied, related to different stages of the disease, and common intake.This is a more visible and detectable symptom. As a result of the deformation of the tripolytics, the blood flow mechanics of the heart changed, and some of the veins were mixed into the artery with insufficient oxygen, resulting in some parts of the patient ‘ s body, such as lips, nail beds, nostrils, etc. This phenomenon tends to be more pronounced after the activity or when the baby is crying, because the body has increased oxygen consumption and the heart is unable to properly perform the oxidation and blood supply, which is further exacerbated.Respiratory difficulties: As the disease develops, when the downward deformation of the three peripals has an impact on the function of the right heart, which leads to an increase in lung silt, the patient starts to feel the aerobics, breathing is hard, and needs to stop and rest for a few minutes to relieve him. This is due to the restriction of the blood function of the heart pump, the inability of the inside of the lungs to effectively cycling and the disruption of the gas exchange function.• Sit-in breathing: even in a state of rest, when the condition is further aggravated, the patient may have difficulty breathing, and a serious patient may even have to take a position at the end (sitting on the side of the bed, leaning under his legs, leaning forward, etc.) to feel a little smoother. This is due to the increased levels of lung silt and respiratory difficulties, which are further exacerbated by the increase in cardiac respiration during flat sleep.On the one hand, heart structure and function change as a result of the downward deformation of the tri-pore, which can lead to cardiac disorders, and on the other hand, the capacity and stress load of the heart change as a result of the development of the disease, such as tremors and pistons. The combination of these factors has led to patients often having an abnormal heart rate and a feeling of panic, and this is often more pronounced after the activity, which further increases the burden on the heart and makes it more visible.Weakness, fatigue due to the impairment of the right heart function, reduced blood flow to the lung and to the body, resulting in inadequate oxygen and nutrient supplies to the air body, and the gradual emergence of signs of overall weakness, fatigue and a marked decline in activity endurance. In the past, for example, routine activities that can be easily accomplished, such as long walking distances, simple housework, etc., can feel exhausted when they become ill.The edema reaches the advanced stage of the disease and, as the condition progresses, there may be signs of right-heart failure, with lower leg edema being more common, first dentible edema in ankles, calves, etc., and then the edema may gradually spread upward to the thigh, or even to the whole body edema, which may be accompanied by cavity fluids such as chest and abdominal water. This is due to the circulation of blood after heart failure, which causes the liquid to seep into the interlocking gap.The specific symptoms of different patients may vary depending on the severity of the downside deformation of the tripolytics and the individual differences, but should the symptoms occur, a detailed medical examination should be conducted in a timely manner.