The cirrhosis of the liver and the prevention of liver cerebral disease: science in life

The cirrhosis of the liver is a chronic liver disease characterized by the replacement of hepatic cells by fibrous tissues after mass necrosis, resulting in significant changes in liver structure and functioning. When hepatic cirrhosis enters the age of non-reparation, the patient is highly likely to combine hepatic cerebral disease, which is a disorder that affects the brain function due to the abnormal exclusion of toxins due to liver dysfunction. Symptoms of liver cerebral disease include, inter alia, memory loss, mental symptoms, hand tremors, and the possibility of a coma in serious cases, which significantly affects the quality of life of patients. However, through scientific lifestyles and medical interventions, we can significantly reduce the risk of liver cerebral disease among patients during cirrhosis.

I. Basic mechanisms for liver cerebral disease

The mechanism for the development of liver cerebral disease is not fully known to date, but its pathological physiological basis is generally considered to be the cleavage between liver cell failure and the cavity vein. When the liver function is impaired, many toxic metabolites from the intestinal tract, such as ammonia, sulfol and short-chain fatty acid, are not detoxified and removed from the liver, but are cycling through a side branch, reaching the brain through a blood and brain barrier, causing a brain dysfunction. The accumulation of these toxic substances in the brain disrupts the brain ‘ s energy metabolism and affects the normal functioning of neurotransmitters, leading to a series of neuropsychiatric symptoms.

II. Adjustments to dietary patterns

1. Low-protein diet: Patients with cirrhosis of the liver should be guided by low-protein, high-calorie dietary principles. In particular, high-protein foods, such as meat and fish, need to be reduced, as high-protein diets can increase the generation of intestinal ammonia, thus increasing the risk of liver cerebral disease. However, aromatic aromatic amino acids in plant proteins are relatively small, with a high number of systolic amino acids, while systolic amino acids can resist some of the toxic substances by disrupting the brain neurofunctional function and can therefore be eaten in small quantities.

2. Maintenance of excretion: Patients with cirrhosis of the liver should be free of excretion one or two times a day to reduce the absorption of intestinal ammonia and other toxins. Fruits, such as bananas, can be eaten, or slow-assulants, such as lactose, can be used to promote intestinal creeping and defecation. 3. Potassium and vitamins: In case of reduced appetite, vomiting or diarrhoea, the cirrhosis of the liver, such as the consumption of fresh cucumber juice, apple juice etc., should be filled in time to avoid low potassium alkalin poisoning, which can lead to the use of ammonia through a blood and brain barrier, which aggravates the liver. At the same time, vitamins and prophylactic bacteria, such as complex vitamins and acidic Bacillus, should be adequately replenished to stabilize the environment in the machine.

4. Avoiding hard foods: In cases of cirrhosis of the liver with edible veins, the consumption of too rough and hard foods, such as fish with thorns, chickens with bones and nuts, should be avoided in order to prevent the scratching of a twisted oesophagus or stomach veins leading to haemorrhage in the upper digestive tract.

III. Combating infection

Patients with cirrhosis are less immune and are vulnerable to infection, which in turn increases the liver burden and increases the risk of liver cerebral disease. Consequently, special attention should be paid to the prevention and treatment of infection among persons with cirrhosis of the liver.

1. Personal hygiene: Patients with cirrhosis of the liver shall maintain their personal hygiene and wash their hands, especially before eating or after touching public facilities. Handwashing should take at least 20 seconds with soap and warm water to reduce the transmission of pathogens.

2. Personal protection: In public places or in close contact with others, persons with cirrhosis of the liver should wear personal protective devices such as masks and gloves to reduce the risk of infection. At the same time, the sharing of household goods, such as utensils and toothbrushes, should be avoided in order to reduce opportunities for transmission of the virus.

3. Periodic medical check-ups: patients with cirrhosis of the liver should undergo periodic liver function check-ups and blood levels tests, as well as medical advice. If anomalies are detected, they should be treated and treated in a timely manner.

Use of conventional drugs

1. Urine: Patients with cirrhosis of the liver are often accompanied by abdominal water, which can help to reduce oedema and abdominal water, but should be administered on medical advice to avoid excessive use leading to electrolyte disorders.

2. Antibiotics: In cases of co-infection, sensitive antibiotics should be selected for treatment based on the results of drug-sensitive tests to prevent infection from increasing the liver burden.

lactating sugar: The oral solution of lactating sugar improves the clinical symptoms of persons with liver cerebral problems by reducing colon pH by increasing the production of organic acid in the colon and reducing the formation and absorption of ammonia.

4. Serial amino acid solution: Serial amino acid solution is a combination formulation with an intravenous infusion that applies to persons with liver cerebral problems. It contains three systolic amino acids that can be metabolized through a blood-brain barrier and a central nervous system, with some neuroprotective effect.

5. Other drugs, such as alumamides, ractrotol, have also helped to reduce the formation and absorption of ammonia and other toxins, thus improving the symptoms of persons with liver cerebral disorders.

Summary

Patients during cirrhosis are highly likely to combine liver cerebral diseases, but through scientific lifestyles and medical interventions we can significantly reduce their risk. In terms of diet, the principles of low protein and high heat should be followed, so as to maintain a smooth flow of poop and the timely filling of potassium and vitamins; in the prevention and treatment of infections, personal hygiene should be maintained, personal protection should be provided and periodic medical examinations should be conducted; and in the treatment of medicines, such as urea, antibiotics, lactose and systolic amino acid solutions should be followed to improve liver function and to mitigate symptoms. Through these measures, we can help patients with cirrhosis to better control their condition and improve their quality of life.