The day before, there was a patient consultation at the online workstation of Professor Wang Cai, an expert in facial convulsions: “My mother has been beating under her right eye for more than a month, without any other discomfort, and has not been resting recently. The doctor says it’s a spasm, giving me mercanamine and Qamasipine, and it’s almost a month away. What causes it?”
Professor Wang Jing: Is it a facial spasm that the patient’s right-hand eye is beating? Is that possible? First, the diagnosis of a fascism cannot be made clear by a month or days of medical history, so it is not done first; second, the symptoms of eyelid tremor are more likely to be due to poor rest, most common perestronic jumps, and some patients also have a perusal, which can take a short period of time, with a shorter three-to-five-day break, with the possibility of a longer period of two to three months or three to five months. If it is true that the beating has taken a long time and the symptoms have not been significantly reduced, it is recommended that a review of the visual examination at the nearest hospital should be undertaken to remove the tumor. At present, patients are paid attention to regulating, regularizing and, above all, regularizing their lives, guaranteeing adequate sleep, rest and diet.
Patient questions: Do you need some nutrition nerve or sleep-improvement medication?
Professor Wang Zing: There is no need, there are fewer drugs to eat, and the best is not to eat.
Patient questions: Does that need to stop the Camassipins and the Mecoamine?
Professor Wang Zing: Yes, we can stop.
Extended reading:
Face twitches often begin with an ecstasy of one side of the eye, which gradually expands to the other side of the side, with the most visible convulsion at the mouth, with very little stress on the abdomen; a lighter first convulsion, which lasts only a few seconds, increases gradually and lasts for several minutes or more; a severe facial convulsion, which leads to the opening of the eye, slanting of the mouth, sometimes irritating and co-mangular limbs, causing walking difficulties; and excessive fatigue and stress can exacerbate the symptoms and gradually cease after sleeping, but the patient is unable to exercise self-control.
In fact, the diagnosis of facial spasms, medical consultations and expression are important. Normally, doctors allow patients to do fast-opening — closed eyes, teeth and unstoppable drumming — in fact, the function of examining five branches of the nerve. If the patient is unable to do so, the corresponding neurological function is problematic. The doctor, in conjunction with the patient ‘ s account of the course of the illness and the symptoms, can generally be inferred from a facial convulsion.
But since 94% of the facial avulsions start with eye-pumping, how do you know if it’s a real avulsions, or just eye-pumps? Time is the best test. Thus, in general, the doctor will inform the patient after three months to review the situation and see if the pumping range has been extended to the mouth, neck, etc.; if the condition progresses quickly within three months, do not wait for three months, and the patient will return to the clinic as soon as possible.
But after three months, is there any way to find out?
The diagnosis of facial spasms relies mainly on doctors to see characteristic clinical manifestations. However, patients still need MRI. Conditional medical units have MRI methods (the 3D-TOF and FIESTA examination sequences) specific to facial avulsions. If this is not the case, an MRI should also be screened for: (i) the purpose of the examination is to: (i) understand whether there are intracircle pathologies, such as tumours, cerebrovascular malformations (AVM), cranial malformations, etc., that lead to relapsing of the face, and (ii) to identify the presence of anatomic vessels in contact with the facial nerve, indicating the type of vascular, the fineness and the degree of pressure on the facial nerve.
In the case of acoustic convulsions, there are two main types of treatment, namely, anti-eclampsia drugs, i.e., Camassipine, Okassipine, Bento Sodium, etc., whose main effect is to reduce the reaction of the central nervous system, while reducing the reaction of the facial nerve, so as to mitigate the symptoms of acoustic convulsions, but it is not possible to make the symptoms disappear, while the second type of drug, which is vitamin B, consists mainly of meta-cobaltamine, vitamins, vitamins B6, B12 and so on, plays a certain nutritional nerve role. As a result, drug treatment is generally not particularly good for motor neurotherapy.
Microvascular repressure (MVD) is the only way to treat the causes of the disease, with a cure rate of over 98 per cent. Advantages are based on a long-term and effective solution to facial pain or convulsion, and maintain aneurological integrity of the nerve, thus maintaining normal neurological functions, altering facial disorders that have emerged after other previous treatments and improving the quality of life of patients.
