The “gold partner” for anti-bacterial drugs and the “prohibition combination”

In the long struggle against disease, anti-bacterial drugs are like loyal health defenders, but they have complex and critical rules for “one-side” or “one-on-one” fighting – “gold partners” who can work hand-in-hand to improve their efficacy, “coercive combinations” that can cause trouble in terms of the safety and efficacy of medicines, and are worth exploring.

Let’s start with the Golden Partners. Beta-neamamine antibacterials (e.g., Amosicillin, capriclo) are combined with beta-neamide inhibitors (e.g., klavic acid, Shubathan) and are a classic combination. Many bacteria are resistant to β-neamide “attacks” that can cause β-neamide to damage the drug structure and render it ineffective. But the enzyme inhibitor can “strike” the enzyme, which is a β-intraamide drug, “sustain the escort”, and the combination of potassium klawite formulations in Amosicillin, which widens the antibacterial spectrum, is powerful against the bacteria that are already resistant, is often used in respiratory, urinary system infections, and makes the treatment more than functional.

Amino sugar (e.g. Quintacin) combined with third-generation cystasy (e.g. cystalcin) is also a good combination for severe grenacosis infections. Amino sugar slurry has a strong virulent effect on the aerobic glyphus, which inhibits the synthesis of bacterial proteins; cystals can destroy the bacterial cell wall, and the two can “shoot out” from different targets, like “twixt” to increase the impact on hard-core bacteria, such as copper-green-fashion cystasy, and play a key role in the treatment of acute pneumonia and complex abdominal infections in hospitals, helping to turn patients into safe people.

However, the antibacterial “coercive combination” hides a crisis. The most alarming is the reuse of the same type of antibacterial drugs. Like two co-uses of bacterium, which appear to be “two-insured”, actually increases the risk of adverse reactions and increases the burden on the liver and kidneys, because of their proximity to anti-bacterial mechanisms, which are not only difficult to co-opt, but also make it easier for the fungus to take advantage of their metabolic disorders to “back-up” and delay conditions, such as the association of afuran with a plaster, contrary to the principle of rational use.

The common use of chlorocin and sulfamide is a typical mix of errors. Both act as a metabolism for bacterial folic acid, in combination with competition for targets, mutual resistance and non-aggression, and may cause severe side effects, such as bone marrow inhibition, allergies, and must not “mix” these two types of drugs in the treatment of infections such as tremor, typhoid and typhoid, in order to prevent them from being mistreated.

Antibacterials and living fungicides are also involved. Live drugs, such as pyrocococcus IV, are often used to regulate intestinal strains and to improve digestion, and, if used at random in the same time as oral antibacterials, antibacterial drugs are “open to kill”, with no distinction between me and the beneficial living bacteria, so that the living fungi agent “does not work”. The correct approach is that the two take 2 – 3 hours apart to ensure their respective efficacy and maintain a micro-ecological balance in the intestinal tract and an orderly process of anti-infection treatment.

Special groups such as the elderly, children, pregnant women, are more cautious in the face of “partnership” and “no taboos”. The reduction of the liver and kidney function of the elderly, the slow metabolism of the drug, the adhesion of the drug with antibacterial drugs, the child ‘ s early organs, the high sensitivity of the drug and its inappropriate combination may affect the growth and development of the child, and the use of the drug during pregnancy, associated with the safety of the mother and the child, can affect the foetus ‘ s bones, the development of the teeth, the prohibition of use during pregnancy, let alone the irrational association with other drugs.

In our daily lives, we get antibacterial drugs that are either “do what you want” or, based on experience, hear what you hear, can be used as a chess game, and lose everything. Doctors must, in accordance with medical instructions, weigh the type of infection, the severity of the disease, the individual ‘ s body, and the combination of well-designed drugs. At the same time, they are actively learning about the use of medications, looking at the “interaction” items of the drug instructions, consulting pharmacists in a timely manner in case of doubt, and not blindly “teams” or “dismantling” antibacterial drugs, so that each drug can be used to best effect and to secure a healthy line of defence.