The killer of a variety of stomach diseases – the fungus of the fungus.
H. pylori is a micro-aerobic grenacobacteria, with a unique spiral structure and urea enzyme activity that enables it to survive in a strong acidic environment in the human stomach. H. Pylori infection is one of the most common chronic bacterial infections worldwide. In my country, it is estimated that about 40 per cent of the population is affected by the disease.
Pathology
Through its viscosity on the surface, the fungus combines with the epipelagic cell of the gastric mucous membrane, and uses urea enzymes to decompose urea to produce ammonia, so that the acidity in the middle and the surrounding environment is adequate for its growth. In addition, H. Pylori is able to circulate a wide range of toxins, such as cytotoxic-related genes A (CagaA) and an empty-bulb toxin A (VacA), which can lead to a stomach mucous reaction and promote functional impairments of the gastric mucous membranes barrier, leading to a range of gastrointestinal diseases.
Means of communication
Sphinx is mainly oral. The bacteria may be found in the mouth of a patient infected with claustrophilia, which may be transmitted to others through, inter alia, saliva, through kissing, co-eating (especially when the same meal is used or not of any kind), graft, parental mouth-to-mouth feeding, etc. In addition, human toothbrushes may also be hidden, further increasing the risk of transmission.
Clinical performance
The clinical behaviour of the claustrophilus infection is diverse, with no symptoms for light people and abdominal pain, abdominal swelling, gas, nausea and vomiting for heavy people. Long-term infections can develop into chronic atrophy, digestive ulcer and even stomach cancer. Some patients may also suffer from other diseases such as lymphoma associated with stomach mucous membranes.
Endoscope feature
Endoscopy is one of the important means of diagnosing the infection of the cholesterol. Typical under-scope manifestations include carbs of the gastric mucous membranes, haemorrhage, oedema, spot bleeding and decomposition. In serious cases, ulcer formation is observed, the edges rise and the base is covered with yellow or white false film. The endoscopy can also be used for rapid urea enzyme testing, tissue dyeing or molecular biology testing to confirm the presence of cholesterocella.
Potential hazards
Sphinococcal infections are closely related to a wide range of gastrointestinal diseases, in particular chronic active stomachitis, digestive ulcer, gastrogen cancer and lymphoma associated with gastric mucous membranes. Among them, cholesterocella infection is considered one of the major risk factors for stomach cancer, which has been classified by the World Health Organization as carcinogen I. In addition, studies have shown that cholesterococcal infections may be associated with other systemic diseases, such as cardiovascular diseases and diseases of the nervous system, but their precise mechanisms need to be further studied and clarified.
Treatment strategy
Standard treatment programmes for cholesterocella infections typically include “quartet therapy” consisting of proton pump inhibitors (PPIs), americium and two antibiotics. The course is usually 10-14 days. Common antibiotic combinations include clacin, Amosilin, Metrazine, etc. After completion of treatment, a non-intrusive 13-C or 14-C exhalation test is recommended to assess the eradication effect. In cases where initial treatment fails, consideration should be given to changing the combination of antibiotics or extending the treatment cycle.
Sphinx infection is a common cause of various gastrointestinal diseases, the diagnosis and treatment of which are subject to the principle of individualization. Early identification and effective eradication of cholesterosomiasis is important for improving the prognosis of patients, especially for the prevention of stomach malignant tumours.
