Proximate spinal disease: a disease that cannot be ignored
It is a major attack on the spinal column and can be chronically inflammable, with varying degrees of stress and argon and the surrounding joints. It is more common among young people and has a slightly higher incidence among men than among women.
Knowledge of the disease is essential for early detection, early treatment and improvement of the quality of life of patients. The causes of direct spinal disease are currently not entirely clear and are generally considered to be closely related to a wide range of genetic, infectious and immune factors.
From a genetic point of view, the human white cell antigen (HLA-B27) is closely linked to the disease of a strong straight spinal column, and most patients are positive. However, not all persons with HLA-B27 positive have an illness, which suggests that environmental factors, such as intestinal or urinary tract infections, may act as a catalyst in their onset. Immunological anomalies are manifested in the wrong attack on the body ‘ s immune system on its own organization, which triggers inflammation in the spinal and joint parts. Its clinical performance has certain characteristics.
Early symptoms tend to be hidden, often with pain in the lower waist or buttocks, stiffness, especially when it is apparent in the morning, and can be mitigated after the event. As the condition progresses, pain and inflexibility gradually spread upward to the spine as a whole, resulting in restrictions on its movement. In serious cases, the spinal column may be so straight that the movement of the patient ‘ s body forwards, back stretches, side bends, etc., is severely hampered, even affecting the respiratory function and body balance. In addition to the spinal column, external ecstasy, such as hips, knee joints, etc., may also be tiring, with symptoms such as joint pain, swelling and mobility disorders.
Some patients may also be associated with eye pathologies, such as iris photophositis, manifested in eye pain, fear, blurred vision, etc. The few suffer from systemic complications such as cardiovascular, lung, etc. The diagnosis of direct spinal disease is based on a combination of the patient ‘ s symptoms, signs, visual examinations and laboratory examinations. X-rays can detect pathological changes to the hips, such as fuzzy joints, bone damage, narrow joint faults, etc.; MRI can detect inflammatory changes to the hips and spines at an earlier stage. In addition to HLA-B27, blood tests also show an increase in inflammation indicators such as blood sunk and C-reactive protein.
In the area of treatment, while it is not possible at present to fully treat straight spinal disease, early diagnosis and standard treatment can effectively control the development of conditions, mitigate symptoms and improve the quality of life of patients. Drug treatment is the main means, including inflammation drugs, which can reduce pain and inflammation rapidly; improved rheumatizers, such as nitrous sulfur, can slow progress; and biological agents work for specific inflammation factors, with significant but relatively high prices.
In addition, in daily life, the patient should focus on functional exercise, maintaining the right standing, sitting and sleeping positions, and regular training in spinal and joint activities to maintain joint activity and muscle strength.
Despite being a more serious disease, direct spinal disease can still lead a normal life as long as patients cooperate actively in treatment and remain under long-term management.
