This walnut clamp is not a walnut clamp — exploring left kidney vulex syndrome.
Definitions
The left renal hysteria syndrome (left renalvein entrapment syndrome), also known as the nutcracker syndrome, NCS, is a disease caused by the oppression of the left renal dystal during the journey into the lower cavity, through the acoustic and intestinal artery, or through the faults between the abdominal and vertebrae, resulting in a series of symptoms, such as blood urine, protein urine, and abdominal pain on the left. Nutcracker phenomenon, NCP refers to the fact that the left kidney vein is constricted between the abdominal aorexus and the upper intestinal membrane and expands its pericardial cavity without causing clinical symptoms. Among them, the left kidney vein is under pressure and with symptoms referred to as NPS and the pressure but not symptoms referred to as NPC.
II. SPECIFIC
Former NCS: Clinically most common, left kidney veins are under pressure between abdominal aorexus and intestinal membranes.
Post NCS: left kidney veins are under pressure between the abdominal aorta and the spine.
Former NCS and later NCS: It is rare for the left kidney vein to repeat malformation, with one branch being pressured between the abdominal aorexus and the intestinal membrane anorexia, and the other branch being pressured between the abdominal aorta and the spinal column. Clinical symptoms
Blood urine: Most commonly, it ranges from under-scope blood to flesh-eye blood, most of which is under-scope blood after evening activity. The main causes are non-renal spherical haemorrhage, which is caused, inter alia, by the obstruction of the return of blood after the pressure on the left renal veins, which results in extended silt, increased stress, irregular traffic in the siltary and renal collection systems, abdominal hysteria in the renal dome, and haemorrhage from the mucous membrane of the renal dome, as well as haemorrhage from the edema.
Pain: The second most common is the pain in the abdominal and left waist, which can be radiationed to the middle of the hip and the back of the thigh, aggravated when sitting, standing, walking or cycling, caused by the high pressure of the left kidney vein, which results in a dyslexic retour disorder and silt.
Direct urine proteins: The specific reasons are not yet clear, and increased cylindrical pressure is considered, resulting in an increase in the filtration of kidney balls, leading to protein urine when the dyspnoea is exceeded.
Reproductive veins.
Male: Transects of the left cumulation to varying degrees.
Women: Pain, urination difficulties, pelvic pain, increased menstruation.
Children and adolescents: The scrotum or left testicles are not well pursued.
Other: Obstruction disorder, chronic fatigue syndrome, and renal veins can be found in serious cases.
Clinical diagnosis
The more commonly accepted diagnostic indicator is that the urine red cell form is a non-renal plume (i.e., the red cell form in the urine is normal over 90%). Calcium excretion in urine is normal (Ca/Cr<0.20). The bladder lens was examined for bleeding from the left urinary tube. Renal examination is normal or mild. The abdominal ultrasound, the CT and the MRI are reflected in the pressure and expansion of the left kidney vein. The lower cavity veins and left renal veins confirm that the left kidney retrenchment is impaired and the left retrenchment is different from the lower cavity pressure at 4 mmHg (normal 1 mmHg, also considered abnormal). To exclude other possible causes of haemorrhagic urine.
Treatment
Conservative treatment: Improved nutrition, avoidance of intense physical activity, sleep-side bedding leads to a mismatch in the angle between SMA and OA, thus reducing the pressure on the left kidney veins and should be periodically reviewed for blood, urine and kidney vascular tops. If the patient has a large amount of protein urine, vascular stressor inhibitors can be used for treatment.
Symptoms of surgical treatment: no significant reduction in symptoms after more than 2 years of observation or internal care; severe non-depressive state or severe male sepsis; serious complications, such as dizziness, anaemia and kidney damage.
Purpose: To relieve left renal veins and to restore the left renal veins and the normal blood flow of their support.
Modes: upper intestinal membrane artery, lower movement of left renal veins – lateral alignment of upper cavity veins, lateral alignment of left renal veins of spleen veins, estuarine nuclei – lower diarrheal dystology, fixed dysentery of left renal veins, etc.
Interventions: left kidney veins can be placed.
VI. Preventive measures
Weight management diet balance: ensure full nutritional intake, reduce high calorie, fat and sugar foods and increase diet fibres.
Motion: Maintain aerobics and appropriate force training, maintain healthy weight and reduce abdominal fat accumulation.
Sports and position: Be careful to avoid violent motion: prevent sudden changes in blood flow mechanics.
(b) Slow change of position: to avoid a mutation of the abdominal aorexus and intestinal membranes affecting blood flow.
(b) Periodic medical examinations: comprehensive medical examinations, including abdominal ultrasound, are conducted periodically.
Enhanced surveillance: Increased frequency and targeting of medical examinations, as recommended by doctors, for those with family history or associated risk factors of disease.
