Treatment of infections with antibacterial drugs

Antibacterial drugs are a powerful weapon for the treatment of infectious diseases, which treat infections in many ways. i. The inhibition of bacterial cell wall synthesis β – Neamide antibiotics: this is a large and commonly used type of antibacterials, including penicillin and headgillin. In the case of penicillin, for example, it can be combined with transcipher enzymes during the synthesis of bacterial cell walls. The main component of the bacterium cell wall is peptide, and in the final stage of the synthesis of peptide, transcipher enzymes play a critical intersection. Penicillin, when combined with transpyridium enzymes, prevents the interlinking of the pelican glucose chain and prevents the normal synthesis of cell walls. This is the case with the construction of houses, where critical connecting parts are missing, resulting in the unsettling of the walls. Owing to the high internal penetration pressure of the bacteria, the bacteria, without the support of a complete cell wall, expand, break and eventually die as a result of overdose. Neptune antibiotics: e.g., vancomycin, mainly used in cell walls of gland positive bacteria. It combines with the D-propamyl-D-propamide end of aluminum precursor in cell wall synthesis, thus preventing the synthesis and interlinking of aluminum. This combination has a high degree of specificity and, like a precise key inserted into a specific lockhole, has blocked critical steps in the construction of a bacterial cell wall. This approach is very effective for the treatment of serious infections caused by drug-resistant bacteria such as methoxysyltin and yellow grapes (MRSA). ii. Antibiotics that affect the synthesis of bacterial proteins Aminocin: like Quincin, Amikane, etc., mainly nuclei of bacteria. Bacteria nuclei are synthetic proteins, and amino-clucose antibiotics can be combined with 30 S-yakis of the nuclei, causing bacteria to make mistakes in synthetic proteins. It leads to the misreading of the mRNA password and to the translation of a sequence of protein amino acids, which produces non-functional proteins. These abnormal proteins do not support normal biological activities of bacteria, such as metabolism, exercise and reproduction, which eventually lead to bacterial deaths. Large ring esters: erythycin, Achmycin, etc. They combine mainly with 50-Syaki of the bacterial nuclei, preventing the extension of the pelican chain. This is like placing a barrier on the water flow line for the production of proteins, which makes it impossible for the protein to be properly assembled. Bacteria are thus unable to synthesize complete and functional proteins and growth and reproduction are inhibited. This inhibition is usually reversible and, when drug concentrations decrease, bacteria may recover. Tetracyclic antibiotics: Tetracyclics, Dossicycline, etc., are mainly combined with the A position on the 30-System of the bacterial nuclei, which prevents aminoylyl-tRNA from entering the A position, thus inhibiting the beginning and extension of the pelican chain. This prevents the normal functioning of the bacteria ‘ protein synthesis machine, and the growth and reproduction of bacteria is contained, to some extent controlling the infection. Interference with bacterial nucleic acid metabolism quinone-like antibiotics: e.g., nofluorinated salsat, left-oxidated salsa. This drug is mainly used for the DNA rotor enzyme (bacterial amphibolase II) and amphibolase IV of bacteria. DNA rotation enzymes play a key role in the reproduction, transfer and repair of bacterial DNA, which is able to decipher the double-helicopter structure of DNA and allow for its successful reproduction and transfer. The combination of quinone-type drugs with DNA rotor enzymes and amphithase IV inhibits their activity, resulting in the normal decomposition of the bacterial DNA superspirator structure, the obstruction of the DNA reproduction and transfer process and the inability of bacteria to reproduce for antibacterial purposes. Li Fuping: mainly for treatment of infections such as tuberculosis. Lifoping is uniquely associated with the bacteria’ RNA polymerase, which is the key enzyme in the bacteria’ transfer process and is responsible for the transfer of genetic information from DNA to mRNA. Lifoping inhibited the activity of the RNA polymerase, which prevented the proper transfer of the MRNA to the bacteria, thus preventing the synthesis of proteins and ultimately inhibiting the growth and reproduction of the bacteria. IV. The structure of the substances affecting bacterial folic acid metabolism sulfamide and methoxymethamphetamine: sulfamide is similar to that of the aminophenate (PABA), for which PABA is required as a feedstock in the process of synthesis of folic acid. Sulfamic drugs can be competitively replaced by PABA, and sulfamide-type drugs are mistakenly combined as PABA by the sulfamide synthetic enzymes involved in folic acid synthesis, thus synthesizing non-activated folic acid analogues and preventing the normal synthesis of folic acid by bacteria. The acetazine, on the other hand, inhibits dihydrofolate reduction enzymes, further preventing bacteria from using folic acid to synthesize tetrahydrofolate. When the two drugs are used together, the metabolism of folic acid in bacteria can be double-cut, producing a concerted antibacterial effect. Antibacterial drugs are used to combat bacteria from multiple angles and to treat infections effectively through these different modes of action. However, in the use of anti-bacterial drugs, medical prescriptions must be strictly followed, rational use must be avoided and drug resistance must be avoided.