Vigilante “Dark Flow”: Discovery drug oesophagusitis

In the pursuit of health, drugs are often our strong helper against disease. However, you may not know that these “decents” who treat people sometimes cause a “storm” in the oeuvre, causing the problem of pharmacological oeuvring. Today, let us lift its mysterious veil and learn how to prevent and respond.

I. Awareness of pharmacological dysentery

Drug-based oesophagusitis, by definition, is the inflammation caused by the injury of the oesophagus. Under normal circumstances, the cuisine is a “pathway” from food and liquids to the stomach, and its mucous membranes have some self-protection mechanisms. However, when certain drugs stay too long inside the duct, or directly irritate or corrosive the duct stick, this balance is broken, resulting in mucous membranes filled with blood, oedema, swollen, and even ulcer formation, which in turn causes a series of symptoms of discomfort, such as pain, difficulty of swallowing. It is not as well known as some major diseases, but it does affect the quality of life of many drug users.

“Blacklist” of pathogenic drugs

Antibiotics: e.g., tetracyclics, Visculin. Such drugs are easily detached in acidic environments and form corrosive substances. When the patient takes the medication, he or she is lying flat, and he or she is likely to remain in the cuisine, especially in the narrow part of the cuisine, in contact with the mucous membrane for long periods of time, and to erode the delicate mucous tissue and cause inflammation. For example, one patient, who was using tetracycline due to respiratory infections, did not drink as prescribed by the doctor and slept quickly after taking the medication, felt stingy when swallowed a few days later and was diagnosed with drug-based oesophagusitis.

Inflammatory drugs: aspirin, Broven and others are commonly used antithermal analgesics, but they may also be “disruptors” of edible mucous membranes. These drugs inhibit the synthesis of prostates, which are protective of the cholesterol mucous membrane, which, when blocked, reduces its resilience to damage and can be easily impaired under drug irritation. People with arthritis who take such drugs for long periods of time are at a higher risk of disease if they do not pay attention to the way they take them.

Duplexate: Medicines such as Sodium Alonthate used to treat osteoporosis are highly irritating for edible mucous membranes. Because of their special requirements, they require large quantities of fresh water to be delivered from empty stomachs and are unable to sleep and eat for half an hour, and if the patient neglects these details, the medication can easily be adhesive to the cuisine mucous membranes, causing serious inflammations and even leading to long-term complications, such as a narrow duct.

Anti-heart disorders: Queenion, for example, has a local anaesthesia, inhibits oesophagus, prolongs the length of time the drug stays in the oesophagus, and increases the risk of mucous membrane damage. A number of heart patients who experience post-brain pain and difficulty swallowing while taking Queenitis need to be alert to the occurrence of pharmacological dysentery.

III. Anatomy of the causes of morbidity

Inadequate drug use: this is one of the most important triggers. Many sufferers are easy to use, drink too little water and cannot quickly enter their stomachs through the ducts; or do so immediately, leading to stagnates within the ducts, prolonged intimacy to the mucous membranes, and the burial of inflammation.

Esophagus per se: The presence of structural anomalies in the cuisine, such as a narrow cuisine, vasectomy, and cylindrosis hinders the passage of drugs through the cuisine, making it easier to accumulate locally and to damage mucous membranes. The weakening of the oesophagus function of older persons has also led to a relative increase in the length of time spent on drugs and a corresponding increase in the risk of disease.

Drug-type and physico-chemical properties: Some pharmaceutical preparations themselves are irritating, such as over-heavy capsules, over-heavy tablets or acidic and alkaline drugs, which can cause direct harm to edible mucous membranes.

IV. Clinical performance

The difficulty of swallowing: When eating food or saliva, the patient has a sense of resistance, which may initially be lighter, only when swallowing solid food, and as the disease increases, it becomes difficult to swallow running food or even saliva, due to edible mucousitis and edema leading to a narrow cavity.

Post-rib pain: The pain is located mostly behind the rib, can burn the pain, sting or invisibility, the pain varies, the pain tends to increase when eating, swallowing and, in some cases, radiation to the back, shoulder, etc., causing great discomfort.

Cardiac, anti-acid: When the oesophate mucous membranes are damaged, the accelerosis of gastric acid is caused, and the patient’s self-conscious chest is scalable, accompanied by an anti-acid and often sour in the mouth.

Nasty blood: When the condition is more serious, the edible mucous membrane is decayed, the ulcer is bleeding, the patient may suffer from vomiting symptoms, which can be fresh red blood or gastrophagus with bloodlines, which is a more critical situation requiring immediate medical attention.

V. Diagnosis and identification

Diagnosis of pharmacological dysentery requires a comprehensive consideration of the patient ‘ s medical history, symptoms and associated examinations. The doctor first asks for details about the type of medication that the patient has recently taken, the dosage, the way in which he/she has been treated, and he/she is highly sceptical if there is a suspected pathogen and if the symptoms are typical of the above. The oesophagus examination is an important means of identifying diseases such as oesophagus, edema, sulphagus, ulcer, etc., which can be directly observed through endoscopy, while other oesophagus diseases, such as oesophagus cancer and oecoma, are also excluded. The edible cavity imaging can also assist in the diagnosis, showing the contours of the edible mucous membrane, the movement and the presence of constrictions. Distinctions are made mainly with anti-fluent cuisine, cuisine cancer, oesophagus disorders, etc., based on the history of the disease, the details of the symptoms and the results of the various examinations.

VI. Prevention and treatment measures

(c) Correct administration of medications: strictly in accordance with medical instructions, take care to take the medications in position, stand or sit as far as possible, deliver them with sufficient water (general recommendation 150 – 200 ml) and ensure that the drugs enter the stomach quickly through the edible tube. Do not immediately lay down after taking a drug, especially on more irritating drugs, such as bihydrate, and remain standing or walking for at least half an hour.

Reasonable drug use: For patients who require long-term administration of drugs that may induce oesophate, such as osteoporosis, the doctor may adjust the programme to reduce the risk of oesopharmaceutical damage on the basis of a medical assessment, such as the introduction of intermittent medicine and the replacement of a drug-type.

Dietary adjustment: During a disease, the diet is light and digestive, avoiding the consumption of spicy, overheated, overheated, overheated, rough foods that exacerbate the mucous lesions of the edible tube. More food is available in the form of permeable or semi-meal, such as rice congee, eggs, vegetable soup, etc., so as to allow adequate rest in the kitchen.

Drug treatment: Once diagnosed with drug-based oesophagusitis, the light person can use mucous membrane protections, such as aluminum sulfur, which can form a protective membrane on the surface of the oesal tube, reduce drug irritation and promote mucous membrane repair; if accompanied by a reaction to gastric acids such as heart fever, antiacid acid and so forth, can use acidic acids, such as Omerazole, Renitidine, etc., to reduce the erosion of gastric acid on the oegic membranes; for patients with apparent pain, painkillers can be adequately mitigated, but care is taken to avoid the use of irritant anal painkillers.

While pharmacological oesophages can be prevented, the key is to raise “safety awareness” of drug use. To understand this knowledge, let’s avoid the “side effects” of the drug-based disease, and guard the food pipes and even the whole body. It is hoped that every reader will keep these points in mind and start a new chapter in science, medicine and a healthy life.

Physicidal tubal inflammation.