What about infectious shock?

Severe infections, particularly in the negative of the Geran, often cause infectious shock. Infective shock, also known as sepsis shock, is a sepsis syndrome shock caused by products such as microorganisms and their toxins. Invasive blood cycles involving micro-organisms in infected stoves, their toxins, cytomas, etc., activate the host’s various cell and body fluid systems; create cytogens and inner-source media, which act on various organs, systems and influence their infusion, leading to ischaemic oxygen, metabolic disorders, functional disorders and even multi-organ function failure. This is an infectious shock. Infective shock is therefore the result of the interaction of microbiological factors and mechanisms for the defence of the body, the amount of toxic force of the microorganisms and the internal environment and response of the organism are important factors in determining the development of infectious shock. Diagnosing sepsis: life-threatening organ dysfunction due to infection-induced host-response disorders. The following two articles should be met at the same time: 2 SOFA rating increased by 2 cents from baseline. A sepsis (infective shock) should be met by the following three conditions: After full liquid recovery, vascularly active drugs are still required to maintain average arterial pressure 65 mmHg. Blood lactate concentration > 2 mmol/L. 2. Infective shock pathological changes. Infective shock reduction in exterior vascular resistance, combined with expansion of the capacity vascular, leads to insufficient effective circulation of blood, resulting in low infusion of tissue organs, and eventually to microcycling disorders. Micro-ring modification is divided into three periods: ischaemic insinuation (early shock), silting insinuation (in shock progress), micro-cycle failure (in shock stress, DIC, irreversible). Periods of emergence do not follow a gradual pattern of development, nor do they have clear boundaries. Functional impairments are characterized by reduced functional cavity vascular density, uneven blood flow distribution and increased microcyclic penetration. 3. Treatment of 1 gas route, correction of low oxygen haematosis, continuous monitoring of blood oxygen saturation and adequate oxygen support. 2 The IVS (liquid resuscitation) principle: In the early years of infectious shock, patients have insufficient blood capacity. The main recommendation is crystall rehydration, with a significant proportion of patients recovering from early liquid recovery. Pay attention to the amount and speed of rehydration when heart failure and kidney failure occur. The vascularly active drugs are still low for full rehydration and require an vascular booster. Dopamine and adrenalin are commonly used drugs, with the preference for deadrenalin (less causing cardiac disorders and stronger vascular contraction). Dopamine is considered only when the risk of heart failure is low and the output is low. Control of infection in search of pathology: bacterology of suspected diseases such as blood testing, fatigue and urine. Antibiotic application: 1h intra-venomal treatment with effective wide spectrum antibacterial drugs after diagnosis. Control of infection stoves: removal of catheters, sepsis, surgery, etc. Note that metabolic acid poisoning is not normally directly granted to sodium carbonate except for kidney failure or ph<7.15. The underlying cause of the acid is under-infusion, and efforts should be made to improve blood flow mechanics indicators. When the capacity is filled, vascularly active drugs are still needed to maintain blood pressure, and there may be relative adrenal cortex deficiencies, supplemented by sugar cortex hormones, as appropriate. Possibilities of sepsis should be considered for cases of organ dysfunction with heat-accumulation organ or for unknown reasons. 5. The drug-based use of sodium Zolacillin is 4.5g + 0.9% NS 100ml. ivgttt q8h Pit test Meropenan 1g + 0.9% NS 100ml ivgtt q8h Cardioactive drug Dopamine (20 mg/2ml/s) Micropumps: Dopamine usage = body weight (kg) x 3, plus 0.9% NS solubility to 50ml. 1 ml/h = 1 μg/(kg.min). The speed of the pump is then the μg/(kg)min. A small dose of 2-5ug/(kg.min) improves kidney blood flow and increases urine. Large dose 5-15ug/(kg.min) improved vascular tension. 5 ml/h pump. 2. The dosage of adrenalin is 0.1-1ug/(kg.min) micropump: (kgX0.3) mg plus 0.9% NS solvent to 50 ml. 1ml/h = 0.1g/(kg.min). It's best to have a central vein.