What about the colored hair mole?

A mosaic hair mole.

Also known as Becker Nevus and Becker.

A chromosomal, polyglytic, and faulty skin tumours, typically characterized by light brown to dark brown, irregular plastered pigmentation, increased hormonal receptor expression and actubated mutation are the main causes of morbidity.

Because of the increase in the larvae and substrate melancholyne particles, the real skin has chromosome cells, so clinically it’s black and dark, and it’s always thought it wasn’t washed.

But the number of melanoid cells is normal, different in nature from freckle moles (skin), tarp moles (skin).

The skin is thicker and the muscular muscular beam is thicker, so it appears to be rough and tenuous over the years.

Becker moles can be classified as hairless and polychaetous, depending on the size of their fur.

Why are you talking about these pathological and clinical characteristics? Because it’s about the choice of treatment!Colored spots tend to merge, slightly thickening and wrinkled.

There is a lack of effective drugs for the treatment of Becker moles and, for two clinical characteristics, the increase in substrate pigments and polychaetes, a variety of phototherapy techniques can be used to blacken and detangle.

Let’s go back to the text of the Guide to the Treatment of Common Diseases Related to Laser Appearance (2024 edition) on the treatment of pigments of hair.

According to the literature, the overall effective rate of color removal is 25 ~80 per cent and the overall effective rate of stripping is 50 ~75 per cent.

Treatment is prone to relapse and requires more treatment.

The parameters, such as energy density, vary according to equipment, and the following examples are referenced in the literature, and it is important to have a good judgement of how to react to the end-point of treatment and to manage the skin before and after treatment.

The “blacking” lasers, represented by a tune Q laser, are primarily for pigments and are based on “selective photothermal effects”. For those who are stubborn, there is a need to try “violent” means such as laser-stamping or stripping lasers.

1. Switch Q lasers

The end-point of the treatment is a skin frost reaction, sometimes with a point of internal bleeding in a needle.

Clinically commonly used are Q switch 694 nm ruby laser, Q switch 755 nm emerald laser, Q switch 1064 nm Nd:YAG laser.

Multiple treatments are usually required, with a 2-3 month interval.

Leather-second lasers are also broad-based Q-modified lasers that are similar to the end-point reactions, spacing times, therapeutic effects, etc., of the pigmentous hair skin mole.

The “blacking” lasers, represented by a tune Q laser, are primarily for pigments and are based on “selective photothermal effects”.

The advantage is that there is little damage to normal surroundings, and the disadvantage is that it is too gentle and the overall effect is not ideal.

Because pigments are not a simple pigmentation problem, especially when the skin is thickened or has a cystal rash, which directly affects the depth of the laser ‘ s effects, it is better to deal with freckles and old-age spots.

For those who are stubborn,

There is a need to try a simpler and more violent approach.


2. Point-forming lasers

As another major development in the modern history of laser development, “the luminous luminous heat” avoids leaving the battlefield piece unattended, but instead preserves a living force around a spot of microinjury, thus recovering faster.

It is often used to improve pox pits, light aging, hairy holes, etc.

During color-based hair-skin mole treatment, the choice is to strip or non-skin point lasers.

The former are stronger, such as Big Brother CO2 pointer lasers and Er-YAG pointer lasers, where the end-point of the treatment is a pointer of a needle, and a little bleeding.

The latter recovered for a shorter period, such as 1550 nm glass-point lasers, with a therapeutic endpoint reaction of skin erythromatoma, a slight increase in color and a four to eight-week interval.

3. Deleting lasers

It’s the traditional lasers used for the gills.

It is not only for melanoid particles, but rather for water, and this photothermal activity is not selective and directly damages local skin tissues.

For example, 2940 nmEr: YAG lasers, the end point of the treatment is completely stripped of the skin and the skin of the skin, with no color residue, with a point of haemorrhage of needles, with a three-month interval of treatment.

Compared to CO2 lasers, it’s light and relatively precise. 4. Decorated lasers

Primarily, some long-pulse-wide lasers are very mature, as long as the number of treatments is sufficient and the effect of defaming is certain.

The end-point of the treatment is slightly red, edema, cystal rash response, slight increase in color.

Currently clinically applied are long-spirited ruby lasers, long-band wide emerald lasers, long-pulse 1064Nd:YAG lasers, 577nm yellow lasers and semiconductor lasers.

5. Strong pulses

It’s black, it’s hair, it’s skin, it’s physical beauty.

The wavelength of the high-pulse filtration filter can be 560, 590, 640, 695 nm, energy density, subpulse count, subpulse width and subpulse spacing vary according to the equipment.

The end-point of the treatment is microred, the color is slightly deepened, and the hair of the cyst is slightly edema.


In summary:

1. Stripping lasers (recommended as 2940 nmEr:YAG lasers) are strong enough to effectively remove color deposition, and the remaining hair requires combined defaming treatment.

2. Strong pulsed light, because of its wide spectrometry, can function both in pigmentation and increased hair, with better effects on both.

Q-button or skin-second lasers have a slight effect on the fall in Becker mole, are prone to relapse and are difficult to select.

4. There has been limited improvement in the saturation of pigmentation by stripping or non-skinned dot lasers, which is not effective for polyaulytes.

5. Other promising treatments, such as large-lighted low-energy lasers, will require further clinical research.

6. As the mechanisms for the onset of the disease become more clear, it is expected that, in the future, drug treatment with more precise target points will be able to address this vexing chromosomal pelvis at source.

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Beck mole.