Anti-infection drugs are a broad group of drugs used to treat various pathogen infections, consisting mainly of the following types:
I. Antibiotics • Beta – intraamide • penicillin: this is one of the first found and applied antibiotics. Penicillin G is mainly used to treat infections caused by soluble streptococcus, pneumonia streptococcus, etc. The mechanism of action is to suppress the synthesis of bacterial cell walls, thereby killing bacteria. However, due to bacterial resistance problems, many bacteria now produce β-neamase to hydrolyte penicillin, so enzyme-resistant penicillin, such as phenolicillin, is also emerging, mainly for the treatment of enzyme-producing golden fungi infections. • Head bacterium: such drugs have a wide spectrum of antibacterials and are highly antibacterial. The first generation of sepsis e.g., sepsis, is used mainly for the treatment of gland positive infections, particularly skin soft tissue infections. As algebra increases, the antibacterial activity of the gelatin cactus is increased, with third-generation cystasin, eclacteria, pneumocococcus, etc., having a good antibacterial effect, often used to treat multiple infections such as respiratory infections and abdominal infections. The fourth generation of sepsis, such as thalamus, has a wider antigen spectrum and has some antigen activity for a variety of resistant bacteria. Carbon cyanide: is the most widely and most antibacterial-activated beta-nimide antibiotics. Medicines such as amphetamine — Spanish, American — can be used to treat serious, multi-drug-resistant infections such as hospital access to sexually transmitted pneumonia, sepsis, etc. However, due to their extensive antibacterial spectrometry and the fact that inappropriate use can easily lead to the production of drug-resistant bacteria, adaptation certificates are strictly available. • Single-ring β – Nemamide: Aminonam represents a drug that has a high level of antibacterial activity in the gland cactus and is stable for the β – Neamide enzymes, and is used mainly for the treatment of infections caused by aerobic gland cactin, especially those who are allergic to penicillin and septactin, and can be used as an alternative for the treatment of abdominal, urinary reproductive tract infections, etc. • Amino sugar slurry, including Quintacolin, Amica, etc. They act as antibacterials mainly by inhibiting the synthesis of bacterial proteins. Such drugs have a strong anti-bacterial activity for gerranes, such as Quincin, which is often used to treat serious bacterial infections such as sepsis, abdominal infections, etc. However, carbamate-type drugs have severe adverse effects such as ear toxicity and renal toxicity and need to be carefully monitored when used. • Large ringed esters: erythrin is an early representative drug and is now commonly used in achicin, caracinin, etc. They are used mainly for the treatment of gland positive bacterial infections and a number of atypical pathogen infections, such as secondary, chlamydia-induced respiratory infections and urinary reproductive tract infections. The mechanism for the operation of the large mercuric esters is the association of the bacterial nuclei 50S, which inhibits protein synthesis. • Four cyclic groups: including tetracycline, dossicycline, Minocycline, etc. They have antibacterial activity in a wide range of bacteria, lekthas, husks, chlamydia, etc. Tetracyclic drugs are mainly used to treat diseases such as haemorrhoids and urin-genital tract infections, but long-term use can result in adverse effects, such as dystrophy and limited use in children and pregnant women. • Quinone: e.g., Nofluorinated, Left Oxygen, Moxican, etc. These drugs are anti-bacterial in virulent and gland positive bacteria and in a number of atypical pathogens. Their mechanism of action is to inhibit bacterial DNA revolving enzymes (bacterial amphiboles II) and/or amphiboles IV and to hinder the reproduction of bacterial DNA. Drugs such as quinone are widely used for the treatment of a wide range of infectious diseases such as genitourinary tract infections, respiratory infections and intestinal infections, but their use in children and pregnant women can affect the development of the bones and requires caution.
Antiviral drugs • Nuclear (acid) analogues: Azurove is a common drug for the treatment of pure herpes virus infection and of pox – with herpes virus, which prevents the synthesis of the virus’s DNA by inhibiting the virus’s DNA polymerase. There is also Ramifdam, which is mainly used to treat hepatitis B virus infections, which can inhibit the retroversion of hepatitis B virus. • Non-nucleotide antiretroviral inhibitors: for example, Nevelapine, which is used primarily for the treatment of HIV infection, is able to combine with the non-dependant part of the HIV-1 antiretroviral enzyme, inhibits the activity of the enzyme and prevents the virus RNA from retrogression into DNA. Protein enzyme inhibitors: for example, Lopinave-Litonave, used for AIDS treatment, can inhibit the activity of HIV proteomics, so that the virus’s precursor protein cannot crack into mature proteins, thus preventing its assembly and release.
Antiflucts • Polyolene: Sex Cycin B is a wide spectrum antiflucose drug with a strong antibacterial activity for many deep fungi, such as pyromococcus, and invisibility. It has increased cellular membrane penetration by combining it with the rhesus steroids on the fungal cell membrane, leading to the death of fungi by leaking inside the cell. However, nephroxin B is more toxic to kidneys and needs to be closely monitored when used. • Selenium: including fluorine, Icraconol, volleyconium, etc. Fluconium is used mainly for the treatment of infections caused by pyromococcus and oscillosis, such as pyromocococcal vaginalitis and oscillosis meningitis. The action mechanism for americium-type drugs is to inhibit the synthesis of facterium steroids in fungal cell membrane, thereby undermining the integrity of fungal membrane. • Precinct: Carpofine, Mikaffin, etc. are representative of this type of drug. They are mainly used for the treatment of haemophilusemia and invasive pyromococcal infections, among others, by inhibiting fungal cell wall beta – (1,
3) – D – Plucose synthesis enzymes that interfere with the synthesis of fungal cell walls.
