It is a common form of post-natural limit or generalized skin skin skin loss, characterized by the loss of melanoid cell function in part of the skin or in the body, resulting in color loss and white spots. The mechanism for the occurrence of the disease is complex and not yet fully clear, but the study considers it relevant to a number of factors. The root causes of the white phoenix disease are described below in terms of self-immunization, mental and neurochemistry, exposure to chemical substances and other factors.
Mental and neurochemical factors
Psychiatry and neurochemical factors are closely linked to the onset of typhoid. Numerous studies have shown that many white phoenix patients experience negative feelings of trauma, excessive stress, depression or depression during the onset of illness or skin damage. These negative emotions can lead to endocrine system disorders, which in turn affect the normal functioning of melanoid cells. In addition, biochemical substances released from the neurological endpoint of the skin may also cause damage to skin melanol cells, resulting in a decrease in melanone.
Exposure to chemical substances
Long-term exposure to certain chemicals may also lead to white fluoride. These chemicals include phenols, benzidines, etc., which may produce toxins to melanoid cells and lead to the loss of pigments. In some occupations, such as the chemical, paint and rubber industries, there is a higher risk of white fluorine in the case of long-term exposure to these chemicals. Therefore, the prevention of white fluents requires that exposure to these harmful chemicals be avoided.
Self-immunisation factor
Self-immunological diseases are also an important cause of white flu. Self-immunological disease is defined as the wrongful attack of an organism ‘ s immune system on its own health cell or tissue, resulting in tissue damage and abnormal functioning. In the case of typhoid patients, the immune system may attack melanoid cells, leading to a decrease or disappearance of melanoma. Such attacks may be guided by a variety of self-immunosuppressive bodies, such as anti-metacell antibodies. In addition, the role of self-immunogenic factors in white tremors is further supported by the combination of other autoimmune diseases, such as thyroid diseases, diabetes and chronic adrenal decomposition.
Other factors
In addition to the above factors, the occurrence of blizzard can be influenced by a number of other factors. For example, bruising, sunlight exposure and some light-sensoring drugs can lead to white flu. Traumas may lead to the destruction of melanoid cells and affect the sterilisation of melanol; sunlight exposure may accelerate the spread of white spots; Some photo-sensitized drugs may interfere with the normal functioning of melanoid cells. In addition, the lack of trace elements may also be associated with the onset of typhoid. Decreased levels of copper or copper blue protein in the blood and skin of patients with white typhoon have led to lower chesamate activity, which in turn affects the metabolism of melanin.
White phoenix disease is not just a white spot of skin, but a complex disease associated with “inner environment” such as human body beige, blood, immunization, etc. If treatment and adjustments are not made in a timely manner, the extent of damage to the environment in the machine will increase, the activity of chesamate will decline and melanoid cells will continue to die. Therefore, the treatment of typhoid winter cannot stop and must be sustained in early detection and early treatment.
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