What you need to know in life: gout and urea.

As people ‘ s standard of living improves and the diet changes, the incidence of the diseases of gout and ureaemia increases each year, seriously affecting the quality of life of patients. It is therefore important to have a better understanding of the knowledge associated with gout and high urea. Uric acid is the end product of human metabolism. Under normal conditions, daily urine produced and excreted in humans is balanced. When this balance is broken, excessive or reduced excretion of ureic acid results in higher levels of urea acid in blood and high levels of urearate. High urea acid haematosis does not necessarily immediately cause symptoms, but is in a state of high urea acid over a long period of time, and the urea acid crystal is deposited in areas such as the joint kidneys, which in turn induces a series of diseases, such as arrh arthritis, gout, etc. In the case of typical symptoms of arrhythmia, sudden arthritis occurs at night or in the early morning, often with severe pains, such as bruises and bruises. Heat and functional impairment. The first part of the body is often the one-sided first toe joint, the big toe toe root. It can also be tiring with ankles, knee wrists, etc., and can be self-restricted, usually for several days or two weeks. But it’s easy to repeat. If arrhythmia does not treat urea acid crystals for a prolonged period of time, it not only affects the appearance and function of the joint but may also cause the disabling of the joint. In addition, high urea acid haematosis and kidneys cause urea acid kidney disease, kidney stones, etc., which can develop into kidney failure in serious cases. There are a number of factors that can lead to high ureaemia and gout, in which dietary factors are important. Over-ingestion of high-eating foods, such as larvae, seafood and red meat soup, can also affect the excretion of urea and promote the production of urea acid by increasing alcohol consumption, especially beer. In addition, metabolic syndromes such as obesity, hypertension, diabetes and high blood resin are closely related to urea acidemia and interact in a vicious circle. The diagnosis of high urea is based on blood urea. In general, the level of abdominal urea acid is higher than 420 μmol per litre for males and 360 μmol per litre for females, which can be diagnosed as high urearate. For the diagnosis of arrhythmia, the detection of urineic crystallization in joint fluids, in addition to typical clinical performance, is a gold standard for diagnosis. The key to the treatment of hyperureaemia and gout is to control blood urea levels, reduce gouts and prevent complications. In terms of lifestyle interventions, there is a need to reduce the intake of high-pretty foods and to increase the intake of diet-rich fibre-rich foods such as fresh vegetables and fruit and whole grains, while at the same time maintaining a strict ban on alcohol. In particular, beer and white wine, drinking more water and the amount of water per day are recommended at more than 2,000 ml to promote urine acid excretion. For obese persons, weight reduction helps to improve the level of urea acid to avoid excessive stress and cold stress, and triggers, for example, the choice of appropriate drugs for drug treatment based on the patient ‘ s specific circumstances. For example, in the acute onset of arrhythmia, the main use is acne anti-inflammation drug, Autumn alphate. Symptoms of cortex pain relief, such as sugar cortex hormones. In intermittent and chronic periods, urea-acid drugs, such as snortol non-bbutamalone, are required. Controls blood urea levels to the desired extent. Long-term regulated treatment and management. It is important to control the progress of diseases and improve the quality of life of patients.