Acute pancreasitis is a disease of internal digestion of pancreas tissues caused by abnormal insulin activation and may cause other organ dysfunctions. Not only does it have a sudden onset of disease, but it can deteriorate rapidly and in serious cases endanger lives. Knowledge and awareness of acute pancreas is therefore crucial. This paper will elaborate on the causes, symptoms, diagnosis, treatment and prevention of acute pancreas.
I. Causes of acute pancreas disease
Acute pancreas is often caused by a number of causes, including, inter alia, cholesterol, alcohol and hyperliptic haemorrhage. Data from China show that these three factors account for over 70 per cent of pancreas.
Cholesterol: Cholesterol is one of the most common causes of acute pancreas. Insulin discharges can be hampered when the cholesterol is sequestered or the pancreas pressurize the lower end of the cholesterol, leading to ingestion of the pancreas tissue itself.
2. Alcohol: Insulin can be damaged by long periods of alcohol consumption, resulting in excessive incubation and increased insulin pressure, leading to acute pancreasitis. In addition, alcohol can directly damage pancreas cells and increase the risk of pancreasitis.
3. High lipid haemorrhagic disease: Plasmodium triester levels in the blood of patients with hyperlipid haemorrhagic disease are too high to accumulate in pancreas, to form a trachea to block incubine and induce acute pancreasitis. In addition, surgical traumas, drug infections, and severe consumption are also contributing factors to acute pancreas. II. Symptoms of acute pancreas
Symptoms of acute pancreas usually include sudden and persistent abdominal pain, as well as associated symptoms of nausea, vomiting, abdominal swelling and fever. Severely ill patients may experience low blood pressure or shock, combined with multiple organ dysfunction, leading to higher mortality rates.
1. Abdominal pain: Abdominal pain is the most common symptom of acute pancreas disease, which occurs suddenly after eating or after extreme fatigue, is located in the upper abdominal centre or left, and continues to increase, with the possibility of radiation to the back or to the hip. Severe abdominal pain may be caused by an oedema or inflammation of an oppressive nerve.
Disgusting, vomiting: Disgusting, vomiting symptoms are high and may begin with food and cholesterol samples, and can lead to vomiting of faeces as the disease deteriorates.
3. Yellow saloon: More common in acute haemorrhagic necrosis of pancreas, probably caused by cholesterol embedding or incubation at the lower end of the general tube.
4. Electrolytic disorders: light pancreasitis may be caused by intestinal palsy and vomiting, while heavy pancreasitis may cause severe dehydration and electrolytic disorders in a short period of time, mainly due to the loss of large quantities of liquids as a result of postperitoneal irritation.
5. Heat: Temperature of light pancreas is within 39°C and can decline after 3-5°C, while the body temperature of heavy pancreas is at 39 – 40°C and may be associated with haemoemia.
Diagnosis of acute pancreas
The diagnosis of acute pancreas is based on symptoms, haematological examinations and visual tests.
Symptoms: Persistence, severe pain in the upper abdominal, and attendant symptoms of nausea, vomiting, etc.
Hematological examination: serosol and fat enzyme are important indicators for the diagnosis of acute pancreas. The general serotonase rises dramatically in hours after the onset of the disease, peaking around 24 hours, and gradually decreases to normal after 4-5 days. It usually rises 24 hours after the onset of the disease, reaches a peak of 48 hours and is normal after 1-2 weeks. Blood fatty enzyme can also be used as a more specific indicator.
3. Visual examinations: abdominal B super, CT and MRI are of great value for the diagnosis of acute pancreas. Insulin swelling, insulin weekly seepage and insulin and pancreatic weekly echoes are detected. The CT is very valuable for acute pancreas diagnosis, for understanding changes in pancreas and conditions around them, and for identifying light and severe pancreas. The abdominal MRI is characterized by the swelling of the pancreas, the disappearance of the bouquets, the knowledge of the cholesterol and the identification of the causes of the diagnosis of pancreas inflammation. IV. Treatment of acute pancreasitis
The treatment of acute pancreas includes, inter alia, general treatment, medication, endoscopy treatment and surgical treatment.
1. General treatment: A medical order of fasting is followed, along with systematic intestine or empty intestine nutrition support. Liquid recovery, maintenance of hydrolysis balance and improved custody are the focus of early treatment.
2. Drug treatment: It usually includes stomach acidics, pain relief, antibacterials, etc. Pancreas can be indirectly suppressed by abdominal acidization, such as Omerazole, Pertolas, Rebelazole, etc. Medicines such as typhus and typhus, which are injected into my body, are painful. Antibacterial drugs are frequently found in quinones, capisculin, carbon methacne, etc.
3. Endoscopy treatment: If diagnosed as cholesterol pancreas, an endoscopy treatment is required within 24 to 72 hours of the onset of the disease, so that the cholesterol can be removed and the chords recovered, and the conditions rapidly improved and the recurrence reduced.
4. Surgery treatment: Patients who are unable to mitigate symptoms or are associated with complications such as obstructive, perforated, abscess, pancreas and infections require early surgical treatment.
V. Prevention of acute pancreasitis
The prevention of acute pancreas includes, inter alia, the reduction of incentives, dietary adjustment, maturation, improved living conditions and disease control.
1. Reduction of incentives: correction of bad habits, prohibition of alcohol consumption, guarantee of diet and prevention of acute pancreas.
2. Dietary regulation: to achieve a reasonable diet, as much as possible, three meals a day, a regular ration and a nutritional balance based on warm and nutritious food.
3. Cotage of effort: avoid overwork or long stayovers that lead to low immunity and can easily induce acute pancreas.
Living conditions: To develop a good lifestyle, to get up early and to get up early, and to increase physical exercise, as appropriate, such as walking or hitting Tai Chi, to help strengthen physical resistance.
5. Disease control: If a person suffers from a chronic disease, such as hyperlipid or diabetes, hemomal sugar and blood pressure should be actively controlled in order to prevent acute pancreas.
In short, acute pancreas is a serious and complex disease that requires great attention. Knowledge of their causes, symptoms, diagnosis, treatment and prevention help to better understand and respond to the disease and protect their health. In our daily lives, we should actively take preventive measures to reduce the incentives for acute pancreas and to improve health.
