In our daily lives, we may occasionally hear the word “yellow”, which is often associated with skin and yellow eyes. What the hell is Huang? Why is it here? What’s your health? Let us explore in depth the secrets behind this physical phenomenon.
I. The metabolic journey of chlamydia
The first thing to understand is the metabolic process of chlamydia. Chlamydia is mainly the result of the destruction of the aging red cells. Red cells survive around 120 days in the blood cycle, and when they age, they are identified and swallowed up by a single nucleus system (mainly spleen, liver and bone marrow). In the process, hemoglobins in the erythrocyte were decomposed, and hemoglobins were gradually converted to chlamydia through a series of enzymes.
This newly produced chlamyrin is known as uncombined chlamydia (indirect chlamyrin), which is not soluble in water and cannot be removed directly through the kidney. Unaccompanied chlamydia enters the liver with blood circulation. In liver cells, a complex set of chemical reactions, mainly as a result of glucose algebrate transfer enzymes, are not combined with cholesterone and glucose alcrylate, which form a combination of chlamydia (direct chlamydia). Combining cholesterone is water soluble and can be discharged into the intestinal tract through the cholesterol system.
In the intestinal tract, the combination of cholesterone, with the effect of intestinal bacteria, undergoes a series of transformations, in which part of the faeces are excreted out of the faeces, and the other part is reabsorbed in the intestinal tract, returning to the liver through the door veins and participating again in the metabolic cycle of chlamor, a process known as chlamyrin ‘ s intestinal liver. Under normal conditions, cholesterol generation, ingestion, integration, excretion and intestinal liver cycling are in a dynamic equilibrium, serocide erythrobin levels are maintained in normal range and no yellow slurry occurs in the body.
II. Categorization and causes of yellow
When there is a failure of a part of the cholesterol metabolism, cholesterol accumulates in the body, resulting in an increase in seroacin levels, which causes yellow sulfide. The yellow sluice can normally be divided into three categories:
1. Soluble yellow. Soluble yellow is caused by the extensive destruction of erythrocytes and the excessive production of cholesterol, which exceeds the capacity of the liver to process it. Common causes include a blood transfusion that is not consistent with the blood type, such as the mistransmitting of type A blood to type B blood patients, which causes a severe soluble reaction; the high incidence of neonatal solubility, especially when the mother and the child have a different blood type, where antibodies in the mother enter the foetus through placenta and attack the red cells of the foetus, resulting in soluble blood; the potential for immunosolation of certain drugs such as penicillin and sulfamide-type drugs; and other self-immunological diseases, such as self-immunolysis anaemia, which produces antibodies against their own red cells in the patient, causing continuous damage to them.
In soluble yellows, large amounts of erythrocyte fractures release large amounts of unaccompanied chlamydia, which is still unable to be fully metabolized in the liver, despite efforts to treat it, resulting in increased chlamydia in the serum. Patients are often shown in mild yellow yellow, with light lemon yellow for skin and membranes. At the same time, there are also symptoms of anaemia such as dizziness, inactivity, panic and spleen swelling due to excessive red cell damage, as spleen is one of the main sites for the destruction of aging red cells, and long-term solubility increases spleen solvency.
Hepatic hysteria Hepatic cystal yellow is the result of hepatocellular damage and disorders in the ingestion, integration and excretion of cholesterone. The various viral hepatitiss (e.g., hepatitis A, B, C, etc.) are common causes of hepatic cytology. When the virus is infected with hepatocellular cells, it causes hepatic inflammation, necrosis and affects normal metabolism of chlamydia. The cirrhosis of the liver is also an important factor, with long-term liver damage resulting in fibrosis of the liver, structural damage and gradual loss of liver cell function. In addition, hepatitis, such as drug poisoning (some antibiotics, anti-tuberculosis, etc.), chemical poisoning (alcohol, carbon tetrachloride, etc.) can damage hepatic cells.
When hepatic cells are damaged, on the one hand, there is a decrease in the ingestion and integration of chlamyrin, resulting in an increase in the blood of chlamydia, and, on the other hand, the already integrated chlamyrin is also elevated by the reverse flow into the blood due to hepatic cell excretion disorder. As a result, hepatocellular serotonin is elevated in those who do not combine chlamydia and chlamydia. In clinical terms, in addition to yellow blubber, patients suffer from symptoms of hepatic impairments such as lack of strength, appetite, nausea, vomiting, liver pain and, in serious cases, complications such as abdominal and liver cerebral diseases. Hepatic functional examination shows a significant increase in enzyme indicators, such as e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e., g., g., e., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g.
3. Courage silt is yellow. Courage silt is yellow because it is prevented from excreting, and it is not properly discharged into the intestinal tract and vice versa. It can be divided into cholesterol in the liver and cholesterol in the liver.
Courage siltation in the liver is common in drug-based liver damage (some drugs can affect the cholesterol function of the liver cell or cause corrosive cell damage to the cholesterol), cirrhosis of the original cholesterol (a self-immuno-disease, with major chords to the liver), chordosis of the liver during pregnancy, etc. In vitro cholesterol is mostly caused by cholesterol, cholesterol, pancreas, etc.
When the cholesterol is silted, the cholesterol cannot be successfully excreted, the pressure inside the cholesterol rises, and the cholesterol in the cholesterol is inverted into the blood, resulting in a significant increase in the serotonine combination. Patients usually have dark yellow and even yellow green skins, accompanied by visible skin itching, as a result of the cura salt deposited in the choreography of the skin. Courage does not enter the intestinal tract, and the light colour of the poop becomes light, and the chlamydia in the urine increases, and the chlamydia deepens, as in the case of tea.
III. Diagnosis of Yellow Sing
When yellow slull symptoms are found, a series of detailed examinations are required to determine the type and cause of the slull.
The first is the collection of medical history on whether the patient has a recent history of blood transfusion, hepatitis, drug use, family genetic history, drinking history and pregnancy. This information is important for the determination of the possible causes of yellow sluice.
During the medical examination, the doctor carefully examines the extent and extent of the yellow slunt, the size of the liver and spleen, the physical surface, the absence of pressure pain and the presence of abdominal water, abdominal swelling, etc. For example, hepatic dystrophy may indicate cirrhosis of the liver; cysts may be large and concussion may be associated with cholesterol.
Laboratory examinations are a key part of the diagnosis of yellow sluice. Serum cholesterol determination is essential, and a preliminary determination of the type of yellow scavenging can be made by measuring the level of total cholesterone, direct cholesterone and indirect cholesterone. In general, if the total cholesterol rises, dominated by indirect cholesterol, more consideration is given to soluble blubber; if both the direct cholercin and the indirect cholesterol rise, hepatocellular blubber is indicated; and if the increase is dominated by direct cholercin, the tendency is for cholesterol silt. At the same time, hepatic function checks are performed, including enzyme indicators such as e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e.g., e., e.g., e.g., e., e., e.g., e., g., g., g., g., e., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., g., In addition, routine blood tests help to detect anaemia in soluble yellow, condensed blood functions can assess the synthesis function of the liver, and various hepatitis virus markers are used to screen viral hepatitis.
Video-censorship also plays an important role in the diagnosis of jaundice. Ultrasound is a common method that clearly shows the form, structure of the liver, cholesterol, larvae and detects the presence of choreography, stones, tumors, etc. Further CT, MRI or MRCP examinations may be required in cases where ultrasound tests are not clear, which can show more accurately the details of the pathology of the liver and internal cholesterol, and are of significant value for the diagnosis of diseases such as cholesterol cancer and insulin cancer. In the case of difficult cases, hepatopsy may also be required, as well as pathological examinations of liver tissues to determine the pathological properties of liver cells and to determine whether hepatic cirrhosis and pharmaceutical liver damage are present.
IV. THE PRINCIPLES OF THE TREATMENT OF YELLOW THOUGHT
The treatment of yellow sepsis is mainly directed at the cause of the disease, with the aim of restoring normal metabolism of chlamydia, reducing yellow sepsis and protecting liver and other vital organ functions.
For soluble yellow blubber, the focus of treatment is on the control of soluble blood. In the case of drug-induced soluble blood, the relevant drugs should be discontinued immediately; self-immuno-salubrious anaemia may require the use of such drugs as sugar cortex hormones, immunosuppressants, etc. to inhibit immune responses and reduce red cell damage. Blood transfusion treatment may be considered in cases of severe anaemia, but care must be taken to avoid a re-emergence of soluble blood. At the same time, efforts are being made to treat the complications of anaemia, such as iron supplements and blood-producing materials such as folic acid, and to improve the symptoms of anaemia.
The core of the treatment for hepatocyte is the protection of hepatic cells, the promotion of hepatic cell restoration and the improvement of liver function. For viral hepatitis, antiviral treatment is required depending on the type of virus, e.g. hepatitis B can use nucleotide analogues and hepatitis C can use direct antiviral drugs. At the same time, hepatopharmaceuticals, such as licoric acid formulations and water falciparum, are given to mitigate hepatitis. In addition to liver cirrhosis treatment, there is a need to treat complications such as the need for abdominal water to limit sodium salt intake, the use of urinants, and control of protein intake, cleaning of intestinal tracts for persons with liver cirrhosis.
The treatment of cholesterol is dependent on the cause of the obstruction. Courage siltation in the liver can contribute to excretion and reduce the siltation of cholesterol by using drugs such as bear deoxychoric acid. In case of cholesterol-induced cholesterol out of the liver, a stone can be removed from the inside mirror by the reverse insulin cholesterol (ERCP) or by a surgical operation; tumoural diseases such as cholesterol cancer and pancreas cancer require a combination of surgical sterilization, chemotherapy and decomposition, depending on the condition. For the skin itching caused by cholesterol silts, a drug such as Colyacin can be used in combination with cholesterol acid to mitigate itching.
In addition, saloon patients need to be careful to rest during their treatment, to avoid overwork and to ensure nutritional balance in order to facilitate physical recovery. V. PURPOSE OF POSITIONS
Although yellow slugs are a symptom of many diseases, we can prevent certain diseases that cause yellow slugs by some measures.
The prevention of viral hepatitis is one of the keys. Hepatitis B vaccination can be effective in preventing infection with hepatitis B; attention to personal hygiene, such as hand washing, avoiding sharing of personal effects such as toothbrushes and razor blades, can reduce opportunities for transmission of the virus. In the case of hepatitis C, the risk of infection can be reduced by avoiding unnecessary injections, blood transfusions and the use of blood products, and by strictly screening blood donors.
In the use of medicines, care should be taken in the use of drugs that may cause liver damage. Before drugs are used, detailed information should be provided on the adverse effects and care of drugs, strictly in accordance with medical prescriptions and to avoid self-abuse or overdose. In the case of long-term use of a drug, hepatic function checks should be conducted on a regular basis in order to detect early detection of pharmaceutical liver damage and to adjust treatment programmes in a timely manner.
For groups with family history or high-risk factors of cholesterol diseases such as cholesterol, cholesterol, etc., the abdominal ultrasound should be carried out on a regular basis in order to detect the stones at an early stage and take appropriate treatment to prevent cholesterol resistance from causing cholesterol silt.
Maintaining a healthy lifestyle is also important for liver health. Drinking alcohol or abating alcohol in appropriate quantities to avoid alcohol-related liver disease caused by a long-term and large-scale drinking of alcohol; a rational diet, with more food enriched in vitamins, proteins and food fibres, reduced intake of high fat and sugar foods, control of body weight and prevention of fat liver.
Yellow is not just as simple as yellow in the skin. It is an unusual external expression of chromosomal metabolism within the body, with many diseases hidden behind it. Knowledge of yellow stings, including their formation mechanisms, classification, diagnostic methods, treatment principles and preventive measures, helps us to detect anomalies in a timely manner in our daily lives, to value the signals sent by the body, to enable early diagnosis and treatment, and to protect the liver and the whole body. In-depth knowledge of the yellow sting, both among health workers and the general public, is important to help us better cope with this common clinical symptoms and to improve health and quality of life.
Huang
